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Publications by Christopher Michael Acker

Transgenic Expression of the Amyloid-Β Precursor Protein-Intracellular Domain Does Not Induce Alzheimer's Disease–Like Traits in Vivo

PLoS ONE

Multidisciplinary

2010

English

Related publications

Amyloid Precursor Protein Processing and Alzheimer's Disease

Annual Review of Neuroscience

Neuroscience

2011

English

Equimolar Production of Amyloid Β-Protein and Amyloid Precursor Protein Intracellular Domain From Β-Carboxyl-Terminal Fragment by Γ-Secretase

Journal of Biological Chemistry

English

Molecular Determinants and Thermodynamics of the Amyloid Precursor Protein Transmembrane Domain Implicated in Alzheimer's Disease

Journal of Molecular Biology

English

Age-Dependent Impairment of Somatosensory Response in the Amyloid Precursor Protein 23 Transgenic Mouse Model of Alzheimer's Disease

Journal of Neuroscience

Neuroscience

2003

English

The Intracellular Domain of the Β-Amyloid Precursor Protein Is Stabilized by Fe65 and Translocates to the Nucleus in a Notch-Like Manner

Journal of Biological Chemistry

English

A Novel Transgenic Rat Model With a Full Alzheimer's-Like Amyloid Pathology Displays Pre-Plaque Intracellular Amyloid-Β-Associated Cognitive Impairment

Journal of Alzheimer's Disease

English

Proteolytic Processing of Amyloid-Β Precursor Protein by Secretases Does Not Require Cell Surface Transport

Journal of Biological Chemistry

English

Mildronate Improves Cognition and Reduces Amyloid-Β Pathology in Transgenic Alzheimer's Disease Mice

Journal of Neuroscience Research

Molecular Neuroscience

Cellular

2013

English

A Heparin-Binding Domain in the Amyloid Protein Precursor of Alzheimer's Disease Is Involved in the Regulation of Neurite Outgrowth

Journal of Neuroscience

Neuroscience

1994

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by Christopher Michael Acker

Transgenic Expression of the Amyloid-Β Precursor Protein-Intracellular Domain Does Not Induce Alzheimer's Disease–Like Traits in Vivo

Related publications

Amyloid Precursor Protein Processing and Alzheimer's Disease

Equimolar Production of Amyloid Β-Protein and Amyloid Precursor Protein Intracellular Domain From Β-Carboxyl-Terminal Fragment by Γ-Secretase

Molecular Determinants and Thermodynamics of the Amyloid Precursor Protein Transmembrane Domain Implicated in Alzheimer's Disease

Age-Dependent Impairment of Somatosensory Response in the Amyloid Precursor Protein 23 Transgenic Mouse Model of Alzheimer's Disease

The Intracellular Domain of the Β-Amyloid Precursor Protein Is Stabilized by Fe65 and Translocates to the Nucleus in a Notch-Like Manner

A Novel Transgenic Rat Model With a Full Alzheimer's-Like Amyloid Pathology Displays Pre-Plaque Intracellular Amyloid-Β-Associated Cognitive Impairment

Proteolytic Processing of Amyloid-Β Precursor Protein by Secretases Does Not Require Cell Surface Transport

Mildronate Improves Cognition and Reduces Amyloid-Β Pathology in Transgenic Alzheimer's Disease Mice

A Heparin-Binding Domain in the Amyloid Protein Precursor of Alzheimer's Disease Is Involved in the Regulation of Neurite Outgrowth