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Publications by Colin Hockings
Bax Crystal Structures Reveal How BH3 Domains Activate Bax and Nucleate Its Oligomerization to Induce Apoptosis
Cell
Biochemistry
Genetics
Molecular Biology
McL-1 and BCL-xL Sequestration of Bak Confers Differential Resistance to BH3-only Proteins
Cell Death and Differentiation
Cell Biology
Molecular Biology
Related publications
RhoA/Rho Kinase Up-Regulate Bax to Activate a Mitochondrial Death Pathway and Induce Cardiomyocyte Apoptosis
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Analysis of Endogenous Bax Complexes During Apoptosis Using Blue Native PAGE: Implications for Bax Activation and Oligomerization
Biochemical Journal
Biochemistry
Cell Biology
Molecular Biology
BAX Activation: Mutations Near Its Proposed Non-Canonical BH3 Binding Site Reveal Allosteric Changes Controlling Mitochondrial Association
Cell Reports
Biochemistry
Genetics
Molecular Biology
Mutation to Bax Beyond the BH3 Domain Disrupts Interactions With Pro-Survival Proteins and Promotes Apoptosis
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
BARD1 Translocation to Mitochondria Correlates With Bax Oligomerization, Loss of Mitochondrial Membrane Potential, and Apoptosis
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
PUMA- And Bax-Induced Autophagy Contributes to Apoptosis
Cell Death and Differentiation
Cell Biology
Molecular Biology
×Phosphorylation of Bax Ser184by Akt Regulates Its Activity and Apoptosis in Neutrophils
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Cytosolic BAX Can Form Autoinhibited Dimers to Suppress Apoptosis
Cancer Discovery
Oncology
Proapoptotic Protein Bax Heterodimerizes With BCL-2 and Homodimerizes With Bax via a Novel Domain (BH3) Distinct From BH1 and BH2
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology