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Publications by Cyril Berthet
Cdk2 and Cdk4 Activities Are Dispensable for Tumorigenesis Caused by the Loss of P53
Molecular and Cellular Biology
Cell Biology
Molecular Biology
Related publications
Cdk2 and Cdk4 Regulate the Centrosome Cycle and Are Critical Mediators of Centrosome Amplification in P53-Null Cells
Molecular and Cellular Biology
Cell Biology
Molecular Biology
Hematopoiesis Specific Loss of Cdk2 and Cdk4 Results in Increased Erythrocyte Size and Delayed Platelet Recovery Following Stress
Haematologica
Hematology
Roles of the Functional Loss of P53 and Other Genes in Astrocytoma Tumorigenesis and Progression
Neuro-Oncology
Cancer Research
Oncology
Neurology
P53, Apaf-1, Caspase-3, and -9 Are Dispensable for Cdk5 Activation During Cell Death
Cell Death and Differentiation
Cell Biology
Molecular Biology
Pluripotent Cell Division Cycles Are Driven by Ectopic Cdk2, Cyclin a/E and E2F Activities
Oncogene
Cancer Research
Genetics
Molecular Biology
Induced Expression of P16INK4aInhibits Both CDK4- And CDK2-Associated Kinase Activity by Reassortment of Cyclin-CDK-Inhibitor Complexes
Molecular and Cellular Biology
Cell Biology
Molecular Biology
Rescue of Neural Tube Defects in Pax-3-Deficient Embryos by P53 Loss of Function: Implications for Pax-3- Dependent Development and Tumorigenesis
Genes and Development
Genetics
Developmental Biology
Phosphorylation of P53 on Key Serines Is Dispensable for Transcriptional Activation and Apoptosis
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
HBx-upregulated lncRNA UCA1 Promotes Cell Growth and Tumorigenesis by Recruiting EZH2 and Repressing P27Kip1/Cdk2 Signaling
Scientific Reports
Multidisciplinary
