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Publications by D P Frazier

Repression of Dmp1 and Arf Transcription by Anthracyclins: Critical Roles of the NF-κB Subunit P65

Oncogene

English

The Herpes Simplex Virus Type 1 Latency-Associated Transcript Promoter Is Activated Through Ras and Raf by Nerve Growth Factor and Sodium Butyrate in PC12 Cells.

English

Related publications

Distinct Roles of the IκB Kinase Α and Β Subunits in Liberating Nuclear Factor κB (NF-κB) From IκB and in Phosphorylating the P65 Subunit of NF-κB

Journal of Biological Chemistry

English

Fas-Associated Factor-1 Inhibits Nuclear Factor-κB (NF-κB) Activity by Interfering With Nuclear Translocation of the RelA (P65) Subunit of NF-κB

Journal of Biological Chemistry

English

Retraction: Modification of Cysteine Residue in P65 Subunit of Nuclear Factor-κB (NF-κB) by Picroliv Suppresses NF-κB–Regulated Gene Products and Potentiates Apoptosis

Cancer Research

Oncology

2018

English

Peptide-siRNA Nanocomplexes Targeting NF-κB Subunit P65 Suppress Nascent Experimental Arthritis

Journal of Clinical Investigation

Medicine

2014

English

The NF-κB P65 and P50 Homodimer Cooperate With IRF8 to Activate iNOS Transcription

English

The Anti-Inflammatory Sesquiterpene Lactone Helenalin Inhibits the Transcription Factor NF-κB by Directly Targeting P65

Journal of Biological Chemistry

English

Site-Specific Phosphorylation of the P65 Protein Subunit Mediates Selective Gene Expression by Differential NF-κB and RNA Polymerase II Promoter Recruitment

Journal of Biological Chemistry

English

TDP-43 Inhibits NF-κB Activity by Blocking P65 Nuclear Translocation

PLoS ONE

Multidisciplinary

2015

English

Transcription of the RelB Gene Is Regulated by NF-κB

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by D P Frazier

Repression of Dmp1 and Arf Transcription by Anthracyclins: Critical Roles of the NF-κB Subunit P65

The Herpes Simplex Virus Type 1 Latency-Associated Transcript Promoter Is Activated Through Ras and Raf by Nerve Growth Factor and Sodium Butyrate in PC12 Cells.

Related publications

Distinct Roles of the IκB Kinase Α and Β Subunits in Liberating Nuclear Factor κB (NF-κB) From IκB and in Phosphorylating the P65 Subunit of NF-κB

Fas-Associated Factor-1 Inhibits Nuclear Factor-κB (NF-κB) Activity by Interfering With Nuclear Translocation of the RelA (P65) Subunit of NF-κB

Retraction: Modification of Cysteine Residue in P65 Subunit of Nuclear Factor-κB (NF-κB) by Picroliv Suppresses NF-κB–Regulated Gene Products and Potentiates Apoptosis

Peptide-siRNA Nanocomplexes Targeting NF-κB Subunit P65 Suppress Nascent Experimental Arthritis

The NF-κB P65 and P50 Homodimer Cooperate With IRF8 to Activate iNOS Transcription

The Anti-Inflammatory Sesquiterpene Lactone Helenalin Inhibits the Transcription Factor NF-κB by Directly Targeting P65

Site-Specific Phosphorylation of the P65 Protein Subunit Mediates Selective Gene Expression by Differential NF-κB and RNA Polymerase II Promoter Recruitment

TDP-43 Inhibits NF-κB Activity by Blocking P65 Nuclear Translocation

Transcription of the RelB Gene Is Regulated by NF-κB