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Publications by D. Di Vizio

DACH1 Is a Cell Fate Determination Factor That Inhibits Cyclin D1 and Breast Tumor Growth

Molecular and Cellular Biology

Cell Biology

Molecular Biology

2006

English

Cyclin D1 Genetic Heterozygosity Regulates Colonic Epithelial Cell Differentiation and Tumor Number in ApcMin Mice

Molecular and Cellular Biology

Cell Biology

Molecular Biology

2004

English

Related publications

Antisense to Cyclin D1 Inhibits Vascular Endothelial Growth Factor-Stimulated Growth of Vascular Endothelial Cells: Implication of Tumor Vascularization

Clinical Cancer Research

Cancer Research

Oncology

2006

English

DACH1 Inhibits Transforming Growth Factor-Β Signaling Through Binding Smad4

Journal of Biological Chemistry

English

Cyclin D1 Promotes Tumor Cell Invasion and Metastasis by Cytoplasmic Mechanisms

Molecular and Cellular Oncology

Cancer Research

Molecular Medicine

2016

English

Epidermal Growth Factor-Stimulated Human Cervical Cancer Cell Growth Is Associated With EGFR and Cyclin D1 Activation, Independent of COX-2 Expression Levels

International Journal of Oncology

Cancer Research

Oncology

2016

English

Cyclin D1 Inhibits Mitochondrial Activity in B Cells

Cancer Research

Oncology

2011

English

PTGF-beta , a Type Beta Transforming Growth Factor (TGF-beta ) Superfamily Member, Is a P53 Target Gene That Inhibits Tumor Cell Growth via TGF-beta Signaling Pathway

Proceedings of the National Academy of Sciences of the United States of America

Multidisciplinary

2000

English

Cyclosporine Inhibits Growth Through the Activating Transcription Factor/cAMP-responsive Element-Binding Protein Binding Site in the Cyclin D1 Promoter

Journal of Biological Chemistry

English

Overexpression of Cyclin D1 Promotes Tumor Cell Growth and Confers Resistance to Cisplatin-Mediated Apoptosis in an Elastase-Myc Transgene-Expressing Pancreatic Tumor Cell Line

Clinical Cancer Research

Cancer Research

Oncology

2005

English

Transforming Growth Factor Beta Stabilizes P15INK4B Protein, Increases P15INK4B-Cdk4 Complexes, and Inhibits Cyclin D1-Cdk4 Association in Human Mammary Epithelial Cells.

Molecular and Cellular Biology

Cell Biology

Molecular Biology

1997

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by D. Di Vizio

DACH1 Is a Cell Fate Determination Factor That Inhibits Cyclin D1 and Breast Tumor Growth

Cyclin D1 Genetic Heterozygosity Regulates Colonic Epithelial Cell Differentiation and Tumor Number in ApcMin Mice

Related publications

Antisense to Cyclin D1 Inhibits Vascular Endothelial Growth Factor-Stimulated Growth of Vascular Endothelial Cells: Implication of Tumor Vascularization

DACH1 Inhibits Transforming Growth Factor-Β Signaling Through Binding Smad4

Cyclin D1 Promotes Tumor Cell Invasion and Metastasis by Cytoplasmic Mechanisms

Epidermal Growth Factor-Stimulated Human Cervical Cancer Cell Growth Is Associated With EGFR and Cyclin D1 Activation, Independent of COX-2 Expression Levels

Cyclin D1 Inhibits Mitochondrial Activity in B Cells

PTGF-beta , a Type Beta Transforming Growth Factor (TGF-beta ) Superfamily Member, Is a P53 Target Gene That Inhibits Tumor Cell Growth via TGF-beta Signaling Pathway

Cyclosporine Inhibits Growth Through the Activating Transcription Factor/cAMP-responsive Element-Binding Protein Binding Site in the Cyclin D1 Promoter

Overexpression of Cyclin D1 Promotes Tumor Cell Growth and Confers Resistance to Cisplatin-Mediated Apoptosis in an Elastase-Myc Transgene-Expressing Pancreatic Tumor Cell Line

Transforming Growth Factor Beta Stabilizes P15INK4B Protein, Increases P15INK4B-Cdk4 Complexes, and Inhibits Cyclin D1-Cdk4 Association in Human Mammary Epithelial Cells.