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Publications by David A DeWitt
Co-Involvement of Mitochondria and Endoplasmic Reticulum in Regulation of Apoptosis: Changes in Cytochrome C, BCL-2 and Bax in the Hippocampus of Aluminum-Treated Rabbits
Brain Research
Neuroscience
Neurology
Developmental Biology
Molecular Biology
Related publications
Tubeimoside-1 Induces Glioma Apoptosis Through Regulation of Bax/BCL-2 and the ROS/Cytochrome C/Caspase-3 Pathway
OncoTargets and Therapy
Oncology
Pharmacology
The Endoplasmic Reticulum in Apoptosis and Autophagy: Role of the BCL-2 Protein Family
Oncogene
Cancer Research
Genetics
Molecular Biology
Involvement of BNIP1 in Apoptosis and Endoplasmic Reticulum Membrane Fusion
EMBO Journal
Immunology
Molecular Biology
Biochemistry
Microbiology
Neuroscience
Medicine
Genetics
Role of Mitochondria and Endoplasmic Reticulum in Taurine-Deficiency-Mediated Apoptosis
Nutrients
Nutrition
Food Science
Dietetics
Apoptosis in Normal Rat Embryo Tissues During Early Organogenesis: The Possible Involvement of Bax and BCL-2.
Archives of Histology and Cytology
Histology
Bax and Bak Can Localize to the Endoplasmic Reticulum to Initiate Apoptosis
Journal of Cell Biology
Medicine
Cell Biology
Endoplasmic Reticulum Mitochondria Contacts Modulate Apoptosis of Renal Cells and Its Implications in Diabetic Neuropathy
EBioMedicine
Biochemistry
Medicine
Genetics
Molecular Biology
Growth Factors Prevent Changes in BCL-2 and Bax Expression and Neuronal Apoptosis Induced by Nitric Oxide
Cell Death and Differentiation
Cell Biology
Molecular Biology
Lipopolysaccharide-Induced Dental Pulp Cell Apoptosis and the Expression of Bax and BCL-2 in Vitro
Brazilian Journal of Medical and Biological Research
Immunology
Cell Biology
Pharmacology
Biochemistry
Biophysics
Neuroscience
Medicine
Toxicology
Physiology
Pharmaceutics