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Publications by David E. Kang
Presenilin 1 Negatively Regulates Β-Catenin/T Cell Factor/Lymphoid Enhancer Factor-1 Signaling Independently of Β-Amyloid Precursor Protein and Notch Processing
Journal of Cell Biology
Medicine
Cell Biology
Novel Role of RanBP9 in BACE1 Processing of Amyloid Precursor Protein and Amyloid Β Peptide Generation
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Related publications
Neuronal Activity and Secreted Amyloid Β Lead to Altered Amyloid Β Precursor Protein and Presenilin 1 Interactions
Neurobiology of Disease
Neurology
Netrin-1 Interacts With Amyloid Precursor Protein and Regulates Amyloid-Β Production
Cell Death and Differentiation
Cell Biology
Molecular Biology
Suppressor of Fused Negatively Regulates Β-Catenin Signaling
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Intracellular Trafficking of Presenilin 1 Is Regulated by Β-Amyloid Precursor Protein and Phospholipase D1
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Mammalian APH-1 Interacts With Presenilin and Nicastrin and Is Required for Intramembrane Proteolysis of Amyloid-Β Precursor Protein and Notch
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Thyroid Hormone Negatively Regulates the Transcriptional Activity of the Β-Amyloid Precursor Protein Gene
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Loss of Function ofATXN1Increases Amyloid Β-Protein Levels by Potentiating Β-Secretase Processing of Β-Amyloid Precursor Protein
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
PDZK1-interacting Protein 1 (PDZK1IP1) Traps Smad4 Protein and Suppresses Transforming Growth Factor-Β (TGF-β) Signaling
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Notch Signaling Dynamically Regulates Adult Β Cell Proliferation and Maturity
Journal of Clinical Investigation
Medicine