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Publications by Dom Bartis

ResolvinD1 Stimulates Epithelial Wound Repair and Inhibits TGF-β-induced EMT Whilst Reducing Fibroproliferation and Collagen Production

Laboratory Investigation
Forensic MedicinePathologyCell BiologyMolecular Biology
2017English

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HCRP1 Inhibits TGF-β Induced Epithelial-Mesenchymal Transition in Hepatocellular Carcinoma

International Journal of Oncology
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2017English

The Phytochemical Shikonin Stimulates Epithelial-Mesenchymal Transition (EMT) in Skin Wound Healing

Evidence-based Complementary and Alternative Medicine
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2013English

Estramustine Phosphate Inhibits TGF-β-Induced Mouse Macrophage Migration and Urokinase-Type Plasminogen Activator Production

Analytical Cellular Pathology
Molecular MedicineCell BiologyCancer ResearchForensic MedicineMedicinePathology
2018English

TWEAK Enhances TGF-β-induced Epithelial-Mesenchymal Transition in Human Bronchial Epithelial Cells

Respiratory Research
PulmonaryRespiratory Medicine
2015English

TGF-β/SMAD4-Regulated LncRNA-LINP1 Inhibits Epithelial-Mesenchymal Transition in Lung Cancer

International Journal of Biological Sciences
Applied MicrobiologyEvolutionEcologyDevelopmental BiologyCell BiologyMolecular BiologySystematicsBehaviorBiotechnology
2018English

Epithelial Stem Cells and Implications for Wound Repair

Seminars in Cell and Developmental Biology
Developmental BiologyCell Biology
2012English

P2.03-16 TGF-β Induced EMT and Stemness Characteristics Are Associated With Epigenetic Regulation in Lung Cancer

Journal of Thoracic Oncology
MedicineOncologyRespiratory MedicinePulmonary
2018English

Sulforaphane Inhibits TGF-β-induced Epithelial-Mesenchymal Transition of Hepatocellular Carcinoma Cells via the Reactive Oxygen Species-Dependent Pathway

Oncology Reports
MedicineCancer ResearchOncology
2016English

Circular RNA Hsa_circ_0008305 (circPTK2) Inhibits TGF-β-induced Epithelial-Mesenchymal Transition and Metastasis by Controlling TIF1γ in Non-Small Cell Lung Cancer

Molecular Cancer
Cancer ResearchOncologyMolecular Medicine
2018English

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