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Publications by Duo Zheng
MST1 Promotes Apoptosis Through Phosphorylation of Histone H2AX
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
CDK3, Target of miR-4469, Suppresses Breast Cancer Metastasis via Inhibiting WNT/Β-Catenin Pathway
Oncotarget
Oncology
Related publications
Histone H2AX Promotes Neuronal Health by Controlling Mitochondrial Homeostasis
Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
Phosphorylation of Histone H2A Promotes Tumorigenesis
Cancer Discovery
Oncology
Histone Deacetylase Inhibitor Trichostatin a Promotes the Apoptosis of Osteosarcoma Cells Through P53 Signaling Pathway Activation
International Journal of Biological Sciences
Applied Microbiology
Evolution
Ecology
Developmental Biology
Cell Biology
Molecular Biology
Systematics
Behavior
Biotechnology
Histone Deacetylase Inhibitors Valproic Acid and Depsipeptide Sensitize Retinoblastoma Cells to Radiotherapy by Increasing H2AX Phosphorylation and P53 Acetylation-Phosphorylation
International Journal of Oncology
Cancer Research
Oncology
Histone H2AX Is a Mediator of Gastrointestinal Stromal Tumor Cell Apoptosis Following Treatment With Imatinib Mesylate
Cancer Research
Cancer Research
Oncology
TIPRL Inhibits Protein Phosphatase 4 Activity and Promotes H2AX Phosphorylation in the DNA Damage Response
PLoS ONE
Multidisciplinary
Phosphoinositide 3-Kinase/Akt Inhibits MST1-mediated Pro-Apoptotic Signaling Through Phosphorylation of Threonine 120.
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Affected Homologous Chromosome Pairing and Phosphorylation of Testis Specific Histone, H2AX, in Male Meiosis Under FKBP6 Deficiency
Journal of Reproduction and Development
Animal Science
Zoology
FGFR4 Phosphorylates MST1 to Confer Breast Cancer Cells Resistance to MST1/2-dependent Apoptosis
Cell Death and Differentiation
Cell Biology
Molecular Biology
