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Publications by Edward G Stopa
Kaolin-Induced Chronic Hydrocephalus Accelerates Amyloid Deposition and Vascular Disease in Transgenic Rats Expressing High Levels of Human APP
Fluids and Barriers of the CNS
Medicine
Molecular Neuroscience
Neurology
Developmental Neuroscience
Cellular
Multiplicity of Cerebrospinal Fluid Functions: New Challenges in Health and Disease
Cerebrospinal Fluid Research
Related publications
Transgenic Mouse Model of Alzheimer's Disease (AD) Expressing Human Β-Site APP Cleaving Enzyme 1 (BACE1)
Science-Business eXchange
Low Levels of Amyloid-Beta and Its Transporters in Neonatal Rats With and Without Hydrocephalus
Cerebrospinal Fluid Research
X11α Haploinsufficiency Enhances Aβ Amyloid Deposition in Alzheimer's Disease Transgenic Mice
Neurobiology of Disease
Neurology
Development of Transgenic Rats Producing Human Β-Amyloid Precursor Protein as a Model for Alzheimer's Disease: Transgene and Endogenous APP Genes Are Regulated Tissue-Specifically
BMC Neuroscience
Neuroscience
Cellular
Molecular Neuroscience
The Effect of Apolipoprotein E Deficiency on Islet Amyloid Deposition in Human Islet Amyloid Polypeptide Transgenic Mice
Diabetologia
Internal Medicine
Endocrinology
Metabolism
Diabetes
Accelerated Progression of Kaolin-Induced Hydrocephalus in Aquaporin-4-Deficient Mice
Journal of Cerebral Blood Flow and Metabolism
Neurology
Cardiovascular Medicine
Cardiology
Modulation of Γ-Secretase Reduces Β-Amyloid Deposition in a Transgenic Mouse Model of Alzheimer's Disease
Neuron
Neuroscience
Evidence of TAU Pathology in Kaolin-Induced Hydrocephalus Model of the Aged Rat
Cerebrospinal Fluid Research
Reducing ADAMTS-3 Inhibits Amyloid Β Deposition in App Knock-In Mouse
Biological and Pharmaceutical Bulletin
Medicine
Pharmacology
Pharmaceutical Science
