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Publications by Eva Pros
Deep Analysis of Acquired Resistance to FGFR1 Inhibitor Identifies MET and AKT Activation and an Expansion of AKT1 Mutant Cells
Oncotarget
Oncology
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MET or NRAS Amplification Is an Acquired Resistance Mechanism to the Third-Generation EGFR Inhibitor Naquotinib
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A Case of EGFR Mutant Lung Adenocarcinoma That Acquired Resistance to EGFR-tyrosine Kinase Inhibitors With MET Amplification and Epithelial-To-Mesenchymal Transition
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Multiple Mutations and Bypass Mechanisms Can Contribute to Development of Acquired Resistance to MET Inhibitors
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Activation of the Met Kinase Confers Acquired Drug Resistance in FGFR-targeted Lung Cancer Therapy
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Combating Acquired TRK Inhibitor Resistance
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Acquired Resistance to BRAF Inhibition in BRAFV600E Mutant Gliomas
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Overexpression of HMGA1 Promotes Anoikis Resistance and Constitutive Akt Activation in Pancreatic Adenocarcinoma Cells
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X-Linked Inhibitor of Apoptosis Protein Inhibits Apoptosis in Inflammatory Breast Cancer Cells With Acquired Resistance to an ErbB1/2 Tyrosine Kinase Inhibitor
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