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Publications by F. P. Alford

Impaired Non-Esterified Fatty Acid Suppression to Intravenous Glucose During Late Pregnancy Persists Postpartum in Gestational Diabetes: A Dominant Role for Decreased Insulin Secretion Rather Than Insulin Resistance

Diabetologia

English

The 24-Hour Plasma Thyrotrophin Profile

Clinical Science

1972

English

Related publications

Cellular Mechanisms for Insulin Resistance in Normal Pregnancy and Gestational Diabetes

English

Insulin Action and Non-Esterified Fatty Acids

Proceedings of the Nutrition Society

English

Insulin Augmentation of Glucose-Stimulated Insulin Secretion Is Impaired in Insulin-Resistant Humans

English

The Effects of a Low-Dose Intravenous Insulin Infusion Upon Plasma Glucose and Non-Esterified Fatty Acid Levels in Very Obese and Non-Obese Human Subjects

English

The Insulin Response to Glucose Infusion in Gestational Diabetes

English

Glucose-Stimulated Insulin Secretion During ?Remission? Of Juvenile Diabetes

English

The Impact of Insulin Resistance on Proinsulin Secretion in Pregnancy: Hyperproinsulinemia Is Not a Feature of Gestational Diabetes

English

CaV1.2 Rather Than CaV1.3 Is Coupled to Glucose-Stimulated Insulin Secretion in INS-1 832/13 Cells

Journal of Molecular Endocrinology

Endocrinology

Molecular Biology

2008

English

Impairment of Early Insulin Response After Glucose Load, Rather Than Insulin Resistance, Is Responsible for Postprandial Hyperglycemia Seen in Obese Type 2 Diabetes. Assessment Using Nateglinide, a New Insulin Secretagogue.

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by F. P. Alford

Impaired Non-Esterified Fatty Acid Suppression to Intravenous Glucose During Late Pregnancy Persists Postpartum in Gestational Diabetes: A Dominant Role for Decreased Insulin Secretion Rather Than Insulin Resistance

The 24-Hour Plasma Thyrotrophin Profile

Related publications

Cellular Mechanisms for Insulin Resistance in Normal Pregnancy and Gestational Diabetes

Insulin Action and Non-Esterified Fatty Acids

Insulin Augmentation of Glucose-Stimulated Insulin Secretion Is Impaired in Insulin-Resistant Humans

The Effects of a Low-Dose Intravenous Insulin Infusion Upon Plasma Glucose and Non-Esterified Fatty Acid Levels in Very Obese and Non-Obese Human Subjects

The Insulin Response to Glucose Infusion in Gestational Diabetes

Glucose-Stimulated Insulin Secretion During ?Remission? Of Juvenile Diabetes

The Impact of Insulin Resistance on Proinsulin Secretion in Pregnancy: Hyperproinsulinemia Is Not a Feature of Gestational Diabetes

CaV1.2 Rather Than CaV1.3 Is Coupled to Glucose-Stimulated Insulin Secretion in INS-1 832/13 Cells

Impairment of Early Insulin Response After Glucose Load, Rather Than Insulin Resistance, Is Responsible for Postprandial Hyperglycemia Seen in Obese Type 2 Diabetes. Assessment Using Nateglinide, a New Insulin Secretagogue.