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Publications by Florentina Melzow
Comment on “Endothelial Protein C Receptor (EPCR), Protease Activated Receptor-1 (PAR-1) and Their Interplay in Cancer Growth and Metastatic Dissemination” Cancers 2019, 11, 51
Cancers
Cancer Research
Oncology
Related publications
Erratum To: Protease-Activated-Receptor-2 Affects Protease-Activated-Receptor-1-Driven Breast Cancer
Cellular and Molecular Life Sciences
Molecular Neuroscience
Molecular Medicine
Cell Biology
Molecular Biology
Pharmacology
Cellular
Protease-Activated Receptor 1 (PAR1)
Science-Business eXchange
Protease Activated Receptor-1 (PAR-1) Mediated Platelet Aggregation Is Dependant on Clopidogrel Response
Thrombosis Research
Hematology
Matrix Metalloproteinase 1 (MMP1); Protease-Activated Receptor 1 (PAR1)
Science-Business eXchange
Thrombin and Activated Protein C Inhibit the Expression of Secretory Group IIA Phospholipase A2 in the TNF-α-activated Endothelial Cells by EPCR and PAR-1 Dependent Mechanisms
Thrombosis Research
Hematology
Protease-Activated Receptor-1 Supports Locomotor Recovery by Biased Agonist Activated Protein C After Contusive Spinal Cord Injury
PLoS ONE
Multidisciplinary
Protease-Activated Receptor 1-Dependent Neuronal Damage Involves NMDA Receptor Function
Experimental Neurology
Developmental Neuroscience
Neurology
Extracellular Granzyme K Mediates Endothelial Activation Through the Cleavage of Protease-Activated Receptor-1
FEBS Journal
Biochemistry
Cell Biology
Molecular Biology
Potential Importance of Protease Activated Receptor (PAR)-1 Expression in the Tumor Stroma of Non-Small-Cell Lung Cancer
BMC Cancer
Cancer Research
Oncology
Genetics