Discover open access scientific publications

Search, annotate, share and cite publications

Publications by G Lamorte

Silencing of Membrane-Associated Sialidase Neu3 Diminishes Apoptosis Resistance and Triggers Megakaryocytic Differentiation of Chronic Myeloid Leukemic Cells K562 Through the Increase of Ganglioside GM3

Cell Death and Differentiation

Cell Biology

Molecular Biology

2008

English

Related publications

Grandisin Induces Apoptosis in Leukemic K562 Cells

Brazilian Journal of Pharmaceutical Sciences

English

Ganglioside GM3: An Acidic Membrane Component That Increases During Macrophage-Like Cell Differentiation Can Induce Monocytic Differentiation of Human Myeloid and Monocytoid Leukemic Cell Lines HL-60 and U937.

Proceedings of the National Academy of Sciences of the United States of America

Multidisciplinary

1986

English

NEU3 Sialidase Protein Interactors in the Plasma Membrane and in the Endosomes

Journal of Biological Chemistry

English

Induction of Apoptosis and Erythroid Differentiation of Human Chronic Myelogenous Leukemia K562 Cells by Low Concentrations of Lidamycin

English

Activin a Induction of Erythroid Differentiation Sensitizes K562 Chronic Myeloid Leukemia Cells to a Subtoxic Concentration of Imatinib

American Journal of Physiology - Cell Physiology

Physiology

Cell Biology

2014

English

Mobility of Carbohydrate-Containing Structures on the Surfaces Membrane and the Normal Differentiation of Myeloid Leukemic Cells to Macrophages and Granulocytes

Proceedings of the National Academy of Sciences of the United States of America

Multidisciplinary

1973

English

Physiopathological Function of Hematoside (GM3 Ganglioside)

Proceedings of the Japan Academy Series B: Physical and Biological Sciences

English

Inhibition of KPNB1 Inhibits Proliferation and Promotes Apoptosis of Chronic Myeloid Leukemia Cells Through Regulation of E2F1

OncoTargets and Therapy

Oncology

Pharmacology

2019

English

Combination of Fludarabine and Imatinib Induces Apoptosis Synergistically Through Loss of Mitochondrial Membrane Potential and Increases in Caspase-3 Enzyme Activity in Human K562 Chronic Myleloid Leukemia Cells

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by G Lamorte

Silencing of Membrane-Associated Sialidase Neu3 Diminishes Apoptosis Resistance and Triggers Megakaryocytic Differentiation of Chronic Myeloid Leukemic Cells K562 Through the Increase of Ganglioside GM3

Related publications

Grandisin Induces Apoptosis in Leukemic K562 Cells

Ganglioside GM3: An Acidic Membrane Component That Increases During Macrophage-Like Cell Differentiation Can Induce Monocytic Differentiation of Human Myeloid and Monocytoid Leukemic Cell Lines HL-60 and U937.

NEU3 Sialidase Protein Interactors in the Plasma Membrane and in the Endosomes

Induction of Apoptosis and Erythroid Differentiation of Human Chronic Myelogenous Leukemia K562 Cells by Low Concentrations of Lidamycin

Activin a Induction of Erythroid Differentiation Sensitizes K562 Chronic Myeloid Leukemia Cells to a Subtoxic Concentration of Imatinib

Mobility of Carbohydrate-Containing Structures on the Surfaces Membrane and the Normal Differentiation of Myeloid Leukemic Cells to Macrophages and Granulocytes

Physiopathological Function of Hematoside (GM3 Ganglioside)

Inhibition of KPNB1 Inhibits Proliferation and Promotes Apoptosis of Chronic Myeloid Leukemia Cells Through Regulation of E2F1

Combination of Fludarabine and Imatinib Induces Apoptosis Synergistically Through Loss of Mitochondrial Membrane Potential and Increases in Caspase-3 Enzyme Activity in Human K562 Chronic Myleloid Leukemia Cells