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Publications by Gabriella Campadelli-Fiume

Αvβ3-Integrin Regulates PD-L1 Expression and Is Involved in Cancer Immune Evasion

Proceedings of the National Academy of Sciences of the United States of America

Multidisciplinary

2019

English

The Engineering of a Novel Ligand in gH Confers to HSV an Expanded Tropism Independent of gD Activation by Its Receptors

English

A Fully-Virulent Retargeted Oncolytic HSV Armed With IL-12 Elicits Local Immunity and Vaccine Therapy Towards Distant Tumors

English

The Epithelial Αvβ3-Integrin Boosts the MYD88-Dependent TLR2 Signaling in Response to Viral and Bacterial Components

English

Αvβ3 Integrin Boosts the Innate Immune Response Elicited in Epithelial Cells Through Plasma Membrane and Endosomal Toll-Like Receptors

English

Replicative Conditioning of Herpes Simplex Type 1 Virus by Survivin Promoter, Combined to ERBB2 Retargeting, Improves Tumour Cell-Restricted Oncolysis

Scientific Reports

Multidisciplinary

2020

English

oHSV Genome Editing by Means of galK Recombineering

Methods in Molecular Biology

Genetics

Molecular Biology

2019

English

Novel, Soluble Isoform of the Herpes Simplex Virus (HSV) Receptor Nectin1 (Or PRR1-HIgR-HveC) Modulates Positively and Negatively Susceptibility to HSV Infection

English

Coexpression of UL20p and gK Inhibits Cell-Cell Fusion Mediated by Herpes Simplex Virus Glycoproteins gD, gH-gL, and Wild-Type gB or an Endocytosis-Defective gB Mutant and Downmodulates Their Cell Surface Expression

English

Substitution in the Murine Nectin1 Receptor of a Single Conserved Amino Acid at a Position Distal From the Herpes Simplex Virus gD Binding Site Confers High-Affinity Binding to gD

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by Gabriella Campadelli-Fiume

Αvβ3-Integrin Regulates PD-L1 Expression and Is Involved in Cancer Immune Evasion

The Engineering of a Novel Ligand in gH Confers to HSV an Expanded Tropism Independent of gD Activation by Its Receptors

A Fully-Virulent Retargeted Oncolytic HSV Armed With IL-12 Elicits Local Immunity and Vaccine Therapy Towards Distant Tumors

The Epithelial Αvβ3-Integrin Boosts the MYD88-Dependent TLR2 Signaling in Response to Viral and Bacterial Components

Αvβ3 Integrin Boosts the Innate Immune Response Elicited in Epithelial Cells Through Plasma Membrane and Endosomal Toll-Like Receptors

Replicative Conditioning of Herpes Simplex Type 1 Virus by Survivin Promoter, Combined to ERBB2 Retargeting, Improves Tumour Cell-Restricted Oncolysis

oHSV Genome Editing by Means of galK Recombineering

Novel, Soluble Isoform of the Herpes Simplex Virus (HSV) Receptor Nectin1 (Or PRR1-HIgR-HveC) Modulates Positively and Negatively Susceptibility to HSV Infection

Coexpression of UL20p and gK Inhibits Cell-Cell Fusion Mediated by Herpes Simplex Virus Glycoproteins gD, gH-gL, and Wild-Type gB or an Endocytosis-Defective gB Mutant and Downmodulates Their Cell Surface Expression

Substitution in the Murine Nectin1 Receptor of a Single Conserved Amino Acid at a Position Distal From the Herpes Simplex Virus gD Binding Site Confers High-Affinity Binding to gD