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Publications by Gerald M. COHEN

JNK (C-Jun N-Terminal Kinase) and P38 Activation in Receptor-Mediated and Chemically-Induced Apoptosis of T-Cells: Differential Requirements for Caspase Activation

Biochemical Journal

English

Related publications

Activation of C-Jun N-Terminal Kinase (JNK) During Mitosis in Retinal Progenitor Cells

PLoS ONE

Multidisciplinary

2012

English

C-Jun N-Terminal Kinase (JNK)-mediated Induction of mSin1 Expression and mTORC2 Activation in Mesenchymal Cells During Fibrosis

Journal of Biological Chemistry

English

GqProtein-induced Apoptosis Is Mediated by AKT Kinase Inhibition That Leads to Protein Kinase C-Induced C-Jun N-Terminal Kinase Activation

Journal of Biological Chemistry

English

Oxidation-Triggered C-Jun N-Terminal Kinase (JNK) and P38 Mitogen-Activated Protein (MAP) Kinase Pathways for Apoptosis in Human Leukaemic Cells Stimulated by Epigallocatechin-3-Gallate (EGCG): A Distinct Pathway From Those of Chemically Induced and Receptor-Mediated Apoptosis

English

Activation of Stress-Activated Protein Kinase/C-Jun NH2-terminal Kinase and P38 Kinase in Calphostin C-Induced Apoptosis Requires Caspase-3-Like Proteases but Is Dispensable for Cell Death

Journal of Biological Chemistry

English

BRAF; C-Jun N-Terminal Kinase (JNK)

Science-Business eXchange

2013

English

Activation of C-Jun-N-Terminal Kinase Is Required for Apoptosis Triggered by Glutathione Disulfide in Neuroblastoma Cells

Free Radical Biology and Medicine

Biochemistry

Physiology

2005

English

Tumour Necrosis Factor-Induced Activation of C-Jun N-Terminal Kinase Is Sensitive to Caspase-Dependent Modulation While Activation of Mitogen-Activated Protein Kinase (MAPK) or P38 MAPK Is Not

English

Correlation Between Sustained C-Jun N-Terminal Protein Kinase Activation and Apoptosis Induced by Tumor Necrosis Factor-Α in Rat Mesangial Cells

Journal of Biological Chemistry

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by Gerald M. COHEN

JNK (C-Jun N-Terminal Kinase) and P38 Activation in Receptor-Mediated and Chemically-Induced Apoptosis of T-Cells: Differential Requirements for Caspase Activation

Related publications

Activation of C-Jun N-Terminal Kinase (JNK) During Mitosis in Retinal Progenitor Cells

C-Jun N-Terminal Kinase (JNK)-mediated Induction of mSin1 Expression and mTORC2 Activation in Mesenchymal Cells During Fibrosis

GqProtein-induced Apoptosis Is Mediated by AKT Kinase Inhibition That Leads to Protein Kinase C-Induced C-Jun N-Terminal Kinase Activation

Oxidation-Triggered C-Jun N-Terminal Kinase (JNK) and P38 Mitogen-Activated Protein (MAP) Kinase Pathways for Apoptosis in Human Leukaemic Cells Stimulated by Epigallocatechin-3-Gallate (EGCG): A Distinct Pathway From Those of Chemically Induced and Receptor-Mediated Apoptosis

Activation of Stress-Activated Protein Kinase/C-Jun NH2-terminal Kinase and P38 Kinase in Calphostin C-Induced Apoptosis Requires Caspase-3-Like Proteases but Is Dispensable for Cell Death

BRAF; C-Jun N-Terminal Kinase (JNK)

Activation of C-Jun-N-Terminal Kinase Is Required for Apoptosis Triggered by Glutathione Disulfide in Neuroblastoma Cells

Tumour Necrosis Factor-Induced Activation of C-Jun N-Terminal Kinase Is Sensitive to Caspase-Dependent Modulation While Activation of Mitogen-Activated Protein Kinase (MAPK) or P38 MAPK Is Not

Correlation Between Sustained C-Jun N-Terminal Protein Kinase Activation and Apoptosis Induced by Tumor Necrosis Factor-Α in Rat Mesangial Cells