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Publications by Geylani Can
Nilotinib Significantly Induces Apoptosis in Imatinib Resistant K562 Cells With Wild-Type BCR–ABL, as Effectively as in Parental Sensitive Counterparts
Hematology
Hematology
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NPB001-05 Inhibits BCR-Abl Kinase Leading to Apoptosis of Imatinib-Resistant Cells
Frontiers in Bioscience - Elite
Immunology
Molecular Biology
Biochemistry
Microbiology
Medicine
Genetics
Semi‑random Mutagenesis Profile of BCR‑ABL During Imatinib Resistance Acquirement in K562 Cells
Molecular Medicine Reports
Oncology
Genetics
Molecular Biology
Biochemistry
Cancer Research
Molecular Medicine
MPT0B169, a New Antitubulin Agent, Inhibits BCR-Abl Expression and Induces Mitochondrion-Mediated Apoptosis in Nonresistant and Imatinib-Resistant Chronic Myeloid Leukemia Cells
PLoS ONE
Multidisciplinary
In Vivo Imaging of BCR-Abl Overexpressing Tumors With a Radiolabeled Imatinib Analog as an Imaging Surrogate for Imatinib
Journal of Nuclear Medicine
Medicine
Nuclear Medicine
Radiology
Imaging
Grandisin Induces Apoptosis in Leukemic K562 Cells
Brazilian Journal of Pharmaceutical Sciences
Toxicology
Pharmaceutics
Pharmacology
In Vitro and in Vivo Activity of SKI-606, a Novel SRC-Abl Inhibitor, Against Imatinib-Resistant BCR-Abl+ Neoplastic Cells
Cancer Research
Cancer Research
Oncology
Anti-Rheumatic Agent Auranofin Induced Apoptosis in Chronic Myeloid Leukemia Cells Resistant to Imatinib Through Both BCR/Abl-Dependent and -Independent Mechanisms
Oncotarget
Oncology
Arginine Butyrate Downregulates P210 BCR-Abl Expression and Induces Apoptosis in Chronic Myelogenous Leukemia Cells
Leukemia
Cancer Research
Oncology
Anesthesiology
Pain Medicine
Hematology
Imatinib Mesylate Induces Cisplatin Hypersensitivity in BCR-Abl+ Cells by Differential Modulation of P53 Transcriptional and Proapoptotic Activity
Cancer Research
Cancer Research
Oncology
