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Publications by Giulia Pontarollo
Comment on “Endothelial Protein C Receptor (EPCR), Protease Activated Receptor-1 (PAR-1) and Their Interplay in Cancer Growth and Metastatic Dissemination” Cancers 2019, 11, 51
Cancers
Cancer Research
Oncology
The Gut Microbiota in Cardiovascular Disease and Arterial Thrombosis
Microorganisms
Virology
Microbiology
Related publications
Erratum To: Protease-Activated-Receptor-2 Affects Protease-Activated-Receptor-1-Driven Breast Cancer
Cellular and Molecular Life Sciences
Molecular Neuroscience
Molecular Medicine
Cell Biology
Molecular Biology
Pharmacology
Cellular
Protease-Activated Receptor 1 (PAR1)
Science-Business eXchange
Protease Activated Receptor-1 (PAR-1) Mediated Platelet Aggregation Is Dependant on Clopidogrel Response
Thrombosis Research
Hematology
Matrix Metalloproteinase 1 (MMP1); Protease-Activated Receptor 1 (PAR1)
Science-Business eXchange
Thrombin and Activated Protein C Inhibit the Expression of Secretory Group IIA Phospholipase A2 in the TNF-α-activated Endothelial Cells by EPCR and PAR-1 Dependent Mechanisms
Thrombosis Research
Hematology
Protease-Activated Receptor-1 Supports Locomotor Recovery by Biased Agonist Activated Protein C After Contusive Spinal Cord Injury
PLoS ONE
Multidisciplinary
Protease-Activated Receptor 1-Dependent Neuronal Damage Involves NMDA Receptor Function
Experimental Neurology
Developmental Neuroscience
Neurology
Extracellular Granzyme K Mediates Endothelial Activation Through the Cleavage of Protease-Activated Receptor-1
FEBS Journal
Biochemistry
Cell Biology
Molecular Biology
Potential Importance of Protease Activated Receptor (PAR)-1 Expression in the Tumor Stroma of Non-Small-Cell Lung Cancer
BMC Cancer
Cancer Research
Oncology
Genetics