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Publications by Graham R. Williams
Advanced Bone Formation in Mice With a Dominant-Negative Mutation in the Thyroid Hormone Receptor Β Gene Due to Activation of WNT/Β-Catenin Protein Signaling
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
MELAS Syndrome, Diabetes and Thyroid Disease: The Role of Mitochondrial Oxidative Stress
Clinical Endocrinology
Endocrinology
Metabolism
Diabetes
Related publications
WNT/Β-Catenin Signaling Activates Bone Morphogenetic Protein 2 Expression in Osteoblasts
Bone
Endocrinology
Physiology
Histology
Metabolism
Diabetes
Icariin Augments Bone Formation and Reverses the Phenotypes of Osteoprotegerin-Deficient Mice Through the Activation of WNT/Β-Catenin-BMP Signaling
Evidence-based Complementary and Alternative Medicine
Alternative Medicine
Complementary
Bone Morphogenetic Protein-2 Enhances WNT/Β-Catenin Signaling-Induced Osteoprotegerin Expression
Genes to Cells
Medicine
Genetics
Cell Biology
Novel Β-Carbolines Inhibit WNT/Β-Catenin Signaling
Cell Death and Disease
Molecular Neuroscience
Immunology
Cell Biology
Cancer Research
Cellular
Medicine
Withdrawal: Pathogen-Specific TLR2 Protein Activation Programs Macrophages to Induce WNT-Β-Catenin Signaling.
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
A DNA Binding Mutation in Estrogen Receptor-Α Leads to Suppression of WNT Signaling via Β-Catenin Destabilization in Osteoblasts
Journal of Cellular Biochemistry
Biochemistry
Cell Biology
Molecular Biology
Activation of the WNT/Β-Catenin Signaling Pathway Causes Radioresistance in Colon Cancer
International Journal of Radiation Oncology Biology Physics
Oncology
Radiology
Cancer Research
Radiation
Nuclear Medicine
Imaging
Figure 5: MEG3 Inhibited the Activation of WNT/Β-Catenin Signaling in Podocytes.
Nuclear Accumulation of Β-Catenin Protein Indicates Activation of WNT Signaling in Chemically Induced Rat Nephroblastomas
Pediatric and Developmental Pathology
Pediatrics
Perinatology
Forensic Medicine
Medicine
Child Health
Pathology