Discover open access scientific publications

Search, annotate, share and cite publications

Publications by H W Amtoft

Stability of AML1 (Core) Site Enhancer Mutations in T Lymphomas Induced by Attenuated SL3-3 Murine Leukemia Virus Mutants.

Journal of Virology

English

Sequence Tags of Provirus Integration Sites in DNAs of Tumors Induced by the Murine Retrovirus SL3-3.

English

Related publications

Second-Site Proviral Enhancer Alterations in Lymphomas Induced by Enhancer Mutants of SL3-3 Murine Leukemia Virus: Negative Effect of Nuclear Factor 1 Binding Site.

English

Increased Lymphomagenicity and Restored Disease Specificity of AML1 Site (Core) Mutant SL3-3 Murine Leukemia Virus by a Second-Site Enhancer Variant Evolved in Vivo.

English

Mutation of All Runx (AML1/Core) Sites in the Enhancer of T-Lymphomagenic SL3-3 Murine Leukemia Virus Unmasks a Significant Potential for Myeloid Leukemia Induction and Favors Enhancer Evolution Toward Induction of Other Disease Patterns

English

Increased Induction of Osteopetrosis, but Unaltered Lymphomagenicity, by Murine Leukemia Virus SL3-3 After Mutation of a Nuclear Factor 1 Site in the Enhancer

English

Tissue Distribution and Timing of Appearance of Polytropic Envelope Recombinants During Infection With SL3-3 Murine Leukemia Virus or Its Weakly Pathogenic SL3 Myb5 Mutant

English

The Activated Mlvi-4 Locus in Moloney Murine Leukemia Virus-Induced Rat T-Cell Lymphomas Encodes an Env/Mlvi-4 Fusion Protein.

English

A New Class of Murine Leukemia Virus Associated With Development of Spontaneous Lymphomas.

Proceedings of the National Academy of Sciences of the United States of America

Multidisciplinary

1977

English

Ror (Rorc) Is a Common Integration Site in Type B Leukemogenic Virus-Induced T-Cell Lymphomas

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by H W Amtoft

Stability of AML1 (Core) Site Enhancer Mutations in T Lymphomas Induced by Attenuated SL3-3 Murine Leukemia Virus Mutants.

Sequence Tags of Provirus Integration Sites in DNAs of Tumors Induced by the Murine Retrovirus SL3-3.

Related publications

Second-Site Proviral Enhancer Alterations in Lymphomas Induced by Enhancer Mutants of SL3-3 Murine Leukemia Virus: Negative Effect of Nuclear Factor 1 Binding Site.

Increased Lymphomagenicity and Restored Disease Specificity of AML1 Site (Core) Mutant SL3-3 Murine Leukemia Virus by a Second-Site Enhancer Variant Evolved in Vivo.

Mutation of All Runx (AML1/Core) Sites in the Enhancer of T-Lymphomagenic SL3-3 Murine Leukemia Virus Unmasks a Significant Potential for Myeloid Leukemia Induction and Favors Enhancer Evolution Toward Induction of Other Disease Patterns

Increased Induction of Osteopetrosis, but Unaltered Lymphomagenicity, by Murine Leukemia Virus SL3-3 After Mutation of a Nuclear Factor 1 Site in the Enhancer

Tissue Distribution and Timing of Appearance of Polytropic Envelope Recombinants During Infection With SL3-3 Murine Leukemia Virus or Its Weakly Pathogenic SL3 Myb5 Mutant

The Activated Mlvi-4 Locus in Moloney Murine Leukemia Virus-Induced Rat T-Cell Lymphomas Encodes an Env/Mlvi-4 Fusion Protein.

A New Class of Murine Leukemia Virus Associated With Development of Spontaneous Lymphomas.

Ror (Rorc) Is a Common Integration Site in Type B Leukemogenic Virus-Induced T-Cell Lymphomas