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Publications by H. Chin

Ablation of P/Q-Type Ca2+ Channel Currents, Altered Synaptic Transmission, and Progressive Ataxia in Mice Lacking the Alpha 1a-Subunit

Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
1999English

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Enhanced Learning and Memory and Altered GABAergic Synaptic Transmission in Mice Lacking the Α5 Subunit of the GABAAReceptor

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Intact Synaptic GABAergic Inhibition and Altered Neurosteroid Modulation of Thalamic Relay Neurons in Mice Lacking Delta Subunit

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Intrathecal Injection of P/Q Type Voltage-Gated Calcium Channel Antibodies From Paraneoplastic Cerebellar Degeneration Cause Ataxia in Mice

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Frequenin/NCS-1 and the Ca2+-Channel 1-Subunit Co-Regulate Synaptic Transmission and Nerve-Terminal Growth

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Normal Viability and Altered Pharmacokinetics in Mice Lacking Mdr1-Type (Drug-Transporting) P-Glycoproteins

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Altered Short-Term Synaptic Plasticity in Mice Lacking the Metabotropic Glutamate Receptor mGlu7

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2002English

Ca2+ Signaling in Cardiac Myocytes Overexpressing the Alpha1 Subunit of L-Type Ca2+ Channel

Circulation Research
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The Polyglutamine Expansion in Spinocerebellar Ataxia Type 6 Causes a Β Subunit-Specific Enhanced Activation of P/Q-Type Calcium Channels inXenopusOocytes

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Absence of the Γ Subunit of the Skeletal Muscle Dihydropyridine Receptor Increases L-Type Ca2+Currents and Alters Channel Inactivation Properties

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