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Publications by H. Inoko
Combination of HLA-A24, -DQA1*03, and -DR9 Contributes to Acute-Onset and Early Complete -Cell Destruction in Type 1 Diabetes: Longitudinal Study of Residual -Cell Function
Diabetes
Internal Medicine
Endocrinology
Metabolism
Diabetes
Related publications
Capturing Residual Beta Cell Function in Type 1 Diabetes
Diabetologia
Internal Medicine
Endocrinology
Metabolism
Diabetes
HLA-DQA1*1 Contributes to Resistance and A1*3 Confers Susceptibility to Type 1 (Insulin-Dependent) Diabetes Mellitus in Japanese Subjects
Diabetologia
Internal Medicine
Endocrinology
Metabolism
Diabetes
Residual -Cell Function 3-6 Years After Onset of Type 1 Diabetes Reduces Risk of Severe Hypoglycemia in Children and Adolescents
Diabetes Care
Internal Medicine
Endocrinology
Advanced
Metabolism
Specialized Nursing
Diabetes
No Effect of the 1 ,25-Dihydroxyvitamin D3 on -Cell Residual Function and Insulin Requirement in Adults With New-Onset Type 1 Diabetes
Diabetes Care
Internal Medicine
Endocrinology
Advanced
Metabolism
Specialized Nursing
Diabetes
Recognition of HLA Class I-Restricted -Cell Epitopes in Type 1 Diabetes
Diabetes
Internal Medicine
Endocrinology
Metabolism
Diabetes
Residual SS-Cell Function and Microvascular Complications in Type 1 Diabetic Patients
Brazilian Journal of Medical and Biological Research
Immunology
Cell Biology
Pharmacology
Biochemistry
Biophysics
Neuroscience
Medicine
Toxicology
Physiology
Pharmaceutics
The A3 Allele of the HLA-DQA1 Locus Is Associated With Susceptibility to Type 1 Diabetes in Japanese.
Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
Islet‐Immune Interactions in Type 1 Diabetes: The Nexus of Beta Cell Destruction
Clinical and Experimental Immunology
Allergy
Immunology
Can CD44 Be a Mediator of Cell Destruction? The Challenge of Type 1 Diabetes
PLoS ONE
Multidisciplinary
