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Publications by Hella Wobser
Dominant-Negative Suppression of HNF-1α Results in Mitochondrial Dysfunction, INS-1 Cell Apoptosis, and Increased Sensitivity to Ceramide-, but Not to High Glucose-Induced Cell Death
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Related publications
Mitochondrial Dysfunction Links Ceramide Activated HRK Expression and Cell Death
PLoS ONE
Multidisciplinary
Ginsenoside Rg3 Prevents INS-1 Cell Death From Intermittent High Glucose Stress
Islets
Endocrinology
Metabolism
Diabetes
Increased Sensitivity to Cis-Diamminedichloroplatinum Induced Apoptosis With Mitochondrial DNA Depletion
Cell Death and Differentiation
Cell Biology
Molecular Biology
Article Commentary: Increased Mitochondrial Activity in Anthrax-Induced Cell Death
Journal of Cell Death
Biochemistry
Cell Biology
Increased Apoptosis but Not PDX-1 Associated With Increase Glucose Calorie Interval
Diabetes Mellitus
Internal Medicine
Endocrinology
Metabolism
Diabetes
Interleukin-1beta-Induced Alteration in a Beta-Cell Phenotype Can Reduce Cellular Sensitivity to Conditions That Cause Necrosis but Not to Cytokine-Induced Apoptosis
Diabetes
Internal Medicine
Endocrinology
Metabolism
Diabetes
MicroRNA-21, Induced by High Glucose, Modulates Macrophage Apoptosis via Programmed Cell Death 4
Molecular Medicine Reports
Oncology
Genetics
Molecular Biology
Biochemistry
Cancer Research
Molecular Medicine
A High Glucose Condition Sensitizes Human Hepatocytes to Hydrogen Peroxide-Induced Cell Death
Molecular Medicine Reports
Oncology
Genetics
Molecular Biology
Biochemistry
Cancer Research
Molecular Medicine
Suppression of Epithelial Cell Transformation and Induction of Actin Dependent Differentiation by Dominant Negative Rac1, but Not Ras, Rho or Cdc42
Cancer Biology and Therapy
Cancer Research
Oncology
Molecular Medicine
Pharmacology