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Publications by Henrietta Vadászi

Synaptic Mitochondrial Dysfunction and Septin Accumulation Are Linked to Complement-Mediated Synapse Loss in an Alzheimer’s Disease Animal Model

Cellular and Molecular Life Sciences

Molecular Neuroscience

Molecular Medicine

Molecular Biology

English

Related publications

Intracellular Accumulation of Amyloid-Beta – A Predictor for Synaptic Dysfunction and Neuron Loss in Alzheimer’s Disease

Frontiers in Aging Neuroscience

Cognitive Neuroscience

English

Synapse Loss and Progress of Alzheimer’s Disease -A Network Model

Scientific Reports

Multidisciplinary

English

Mitochondrial Abnormalities and Synaptic Loss Underlie Memory Deficits Seen in Mouse Models of Obesity and Alzheimer’s Disease

Journal of Alzheimer's Disease

Clinical Psychology

English

SIRT3 Deregulation Is Linked to Mitochondrial Dysfunction in Alzheimer's Disease

English

Synaptic Dysfunction and Septin Protein Family Members in Neurodegenerative Diseases

Molecular Neurodegeneration

Molecular Neuroscience

Molecular Biology

English

Elevation of Brain Magnesium Prevents Synaptic Loss and Reverses Cognitive Deficits in Alzheimer’s Disease Mouse Model

Molecular Brain

Molecular Neuroscience

Molecular Biology

English

Acetylated Tau in Alzheimer's Disease: An Instigator of Synaptic Dysfunction Underlying Memory Loss

Molecular Biology

English

Mitochondrial Dysfunction in Genetic Animal Models of Parkinson's Disease

Antioxidants and Redox Signaling

Molecular Biology

Clinical Biochemistry

English

Mitochondrial Dysfunction and Alpha-Lipoic Acid: Beneficial or Harmful in Alzheimer’s Disease?

Oxidative Medicine and Cellular Longevity

English