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Publications by Ho-Seong Cho
The Green Tea Component (-)-Epigallocatechin-3-Gallate Sensitizes Primary Endothelial Cells to Arsenite-Induced Apoptosis by Decreasing C-Jun N-Terminal Kinase-Mediated Catalase Activity
PLoS ONE
Multidisciplinary
Association Between Oropharyngeal Microbiome and Weight Gain in Piglets During Pre and Post Weaning Life
Journal of Animal Science and Technology
Ecology
Genetics
Molecular Biology
Biochemistry
Veterinary
Food Science
Animal Science
Zoology
Related publications
GqProtein-induced Apoptosis Is Mediated by AKT Kinase Inhibition That Leads to Protein Kinase C-Induced C-Jun N-Terminal Kinase Activation
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Oxidation-Triggered C-Jun N-Terminal Kinase (JNK) and P38 Mitogen-Activated Protein (MAP) Kinase Pathways for Apoptosis in Human Leukaemic Cells Stimulated by Epigallocatechin-3-Gallate (EGCG): A Distinct Pathway From Those of Chemically Induced and Receptor-Mediated Apoptosis
Biochemical Journal
Biochemistry
Cell Biology
Molecular Biology
Autophagy Activated by the C-Jun N-Terminal Kinase-Mediated Pathway Protects Human Prostate Cancer PC3 Cells From Celecoxib-Induced Apoptosis
Experimental and Therapeutic Medicine
Medicine
Cancer Research
Immunology
Microbiology
Green Tea Polyphenol (−)-Epigallocatechin-3-Gallate Restores Nrf2 Activity and Ameliorates Crescentic Glomerulonephritis
PLoS ONE
Multidisciplinary
Mutually Exclusive Subsets of BH3-Only Proteins Are Activated by the P53 and C-Jun N-Terminal Kinase/C-Jun Signaling Pathways During Cortical Neuron Apoptosis Induced by Arsenite
Molecular and Cellular Biology
Cell Biology
Molecular Biology
Dicoumarol Potentiates Cisplatin-Induced Apoptosis Mediated by C-Jun N-Terminal Kinase in P53 Wild-Type Urogenital Cancer Cell Lines
Oncogene
Cancer Research
Genetics
Molecular Biology
Ricin A-Chain Requires C-Jun N-Terminal Kinase to Induce Apoptosis in Nontransformed Epithelial Cells
International Journal of Biochemistry and Cell Biology
Biochemistry
Cell Biology
MEKK-1, a Component of the Stress (Stress-Activated Protein Kinase/C-Jun N-Terminal Kinase) Pathway, Can Selectively Activate Smad2-Mediated Transcriptional Activation in Endothelial Cells
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Role of Akt and C-Jun N-Terminal Kinase 2 in Apoptosis Induced by Interleukin-4 Deprivation
Molecular Biology of the Cell
Cell Biology
Molecular Biology
