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Publications by Hubert Stangl
Selective Activation of Tumor Necrosis Factor Receptor 2 Induces Anti-Inflammatory Responses and Alleviates Experimental Arthritis
Arthritis and Rheumatology
Rheumatology
Allergy
Immunology
Cervical Vagal Nerve Stimulation Impairs Glucose Tolerance and Suppresses Insulin Release in Conscious Rats
Physiological Reports
Physiology
Related publications
Tumor Necrosis Factor Receptor 2 Signaling Induces Selective C-Iap1-Dependent ASK1 Ubiquitination and Terminates Mitogen-Activated Protein Kinase Signaling
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Selective Improvement of Tumor Necrosis Factor Capture in a Cytokine Hemoadsorption Device Using Immobilized Anti-Tumor Necrosis Factor
Journal of Biomedical Materials Research - Part B Applied Biomaterials
Biomedical Engineering
Biomaterials
A Dual Target-Directed Agent Against Interleukin-6 Receptor and Tumor Necrosis Factor Α Ameliorates Experimental Arthritis
Scientific Reports
Multidisciplinary
Salivary Cortisol and Soluble Tumor Necrosis Factor-Α Receptor II Responses to Multiple Experimental Modalities of Acute Pain
Psychophysiology
Developmental
Cognitive Psychology
Neuropsychology
Educational Psychology
Cognitive Neuroscience
Endocrine
Developmental Neuroscience
Physiological Psychology
Autonomic Systems
Neurology
Neuroscience
Biological Psychiatry
Physiology
Experimental
Tumor Necrosis Factor (TNF) Receptor 1 Signaling Downstream of TNF Receptor-Associated Factor 2
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
The Tumor Necrosis Factor Receptor 2 Signal Transducers TRAF2 and C-Iap1 Are Components of the Tumor Necrosis Factor Receptor 1 Signaling Complex
Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
Role of Tumor Necrosis Factor (TNF) Receptor-Associated Factor 2 (TRAF2) in Distinct and Overlapping CD40 and TNF Receptor 2/CD120b-mediated B Lymphocyte Activation
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Tumor Necrosis Factor-Α Induces Transcriptional Activation of Nuclear Factor-κB in Insulin-Producing Β-Cells
Experimental and Therapeutic Medicine
Medicine
Cancer Research
Immunology
Microbiology
Hydrogen Peroxide Signaling Through Tumor Necrosis Factor Receptor 1 Leads to Selective Activation of C-Jun N-Terminal Kinase
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology