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Publications by Hye-jin You

Endocytosis of the Type III Transforming Growth Factor-Β (TGF-β) Receptor Through the Clathrin-Independent/Lipid Raft Pathway Regulates TGF-β Signaling and Receptor Down-Regulation

Journal of Biological Chemistry

English

Related publications

Caveolin-1 Regulates Transforming Growth Factor (TGF)-β/SMAD Signaling Through an Interaction With the TGF-β Type I Receptor

Journal of Biological Chemistry

English

TMED10 Protein Interferes With Transforming Growth Factor (TGF)-β Signaling by Disrupting TGF-β Receptor Complex Formation

Journal of Biological Chemistry

English

NM23-H1 Tumor Suppressor Physically Interacts With Serine-Threonine Kinase Receptor-Associated Protein, a Transforming Growth Factor-Β (TGF-β) Receptor-Interacting Protein, and Negatively Regulates TGF-β Signaling

Journal of Biological Chemistry

English

In the Absence of Type III Receptor, the Transforming Growth Factor (TGF)-β Type II-B Receptor Requires the Type I Receptor to Bind TGF-β2

Journal of Biological Chemistry

English

TACE-Mediated Ectodomain Shedding of the Type I TGF-β Receptor Downregulates TGF-β Signaling

English

The Type III TGF-β Receptor Suppresses Breast Cancer Progression

Journal of Clinical Investigation

Medicine

2007

English

CD8 Co-Receptor Promotes Susceptibility of CD8+ T Cells to Transforming Growth Factor-Β (TGF-β)-mediated Suppression

Cancer Immunology, Immunotherapy

English

Signaling Mechanisms of Transforming Growth Factor-Β (TGF-β) in Cancer: TGF-β Induces Apoptosis in Lung Cells by a Smad-Dependent Mechanism

2012

English

Modulation of the Functional Binding Sites for TGF-β on the Type II Receptor Leads to Suppression of TGF-β Signaling

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by Hye-jin You

Endocytosis of the Type III Transforming Growth Factor-Β (TGF-β) Receptor Through the Clathrin-Independent/Lipid Raft Pathway Regulates TGF-β Signaling and Receptor Down-Regulation

Related publications

Caveolin-1 Regulates Transforming Growth Factor (TGF)-β/SMAD Signaling Through an Interaction With the TGF-β Type I Receptor

TMED10 Protein Interferes With Transforming Growth Factor (TGF)-β Signaling by Disrupting TGF-β Receptor Complex Formation

NM23-H1 Tumor Suppressor Physically Interacts With Serine-Threonine Kinase Receptor-Associated Protein, a Transforming Growth Factor-Β (TGF-β) Receptor-Interacting Protein, and Negatively Regulates TGF-β Signaling

In the Absence of Type III Receptor, the Transforming Growth Factor (TGF)-β Type II-B Receptor Requires the Type I Receptor to Bind TGF-β2

TACE-Mediated Ectodomain Shedding of the Type I TGF-β Receptor Downregulates TGF-β Signaling

The Type III TGF-β Receptor Suppresses Breast Cancer Progression

CD8 Co-Receptor Promotes Susceptibility of CD8+ T Cells to Transforming Growth Factor-Β (TGF-β)-mediated Suppression

Signaling Mechanisms of Transforming Growth Factor-Β (TGF-β) in Cancer: TGF-β Induces Apoptosis in Lung Cells by a Smad-Dependent Mechanism

Modulation of the Functional Binding Sites for TGF-β on the Type II Receptor Leads to Suppression of TGF-β Signaling