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Publications by I. B. Appelskog
The Novel Imidazoline Compound BL11282 Potentiates Glucose-Induced Insulin Secretion in Pancreatic -Cells in the Absence of Modulation of KATP Channel Activity
Diabetes
Internal Medicine
Endocrinology
Metabolism
Diabetes
Related publications
Novel I1-Imidazoline Agonist S43126 Augment Insulin Secretion in Min6 Cells
Journal of Diabetes & Metabolism
Glycated Albumin Suppresses Glucose-Induced Insulin Secretion by Impairing Glucose Metabolism in Rat Pancreatic Β-Cells
Nutrition and Metabolism
Nutrition
Endocrinology
Dietetics
Medicine
Metabolism
Diabetes
Allantoin Activates Imidazoline I-3 Receptors to Enhance Insulin Secretion in Pancreatic Β-Cells
Nutrition and Metabolism
Nutrition
Endocrinology
Dietetics
Medicine
Metabolism
Diabetes
Leucine Regulation of Glucokinase and ATP Synthase Sensitizes Glucose-Induced Insulin Secretion in Pancreatic -Cells
Diabetes
Internal Medicine
Endocrinology
Metabolism
Diabetes
Absence of SHB Impairs Insulin Secretion by Elevated FAK Activity in Pancreatic Islets
Journal of Endocrinology
Endocrinology
Metabolism
Diabetes
Loss of LAMTOR1 in Pancreatic Β‑cells Increases Glucose‑stimulated Insulin Secretion in Mice
International Journal of Molecular Medicine
Medicine
Genetics
Mechanisms of Glucose-Induced Secretion of Pancreatic-Derived Factor (PANDER or FAM3B) in Pancreatic -Cells
Diabetes
Internal Medicine
Endocrinology
Metabolism
Diabetes
Glucagon‑like Peptide‑1 Potentiates Glucose‑stimulated Insulin Secretion via the Transient Receptor Potential Melastatin 2 Channel
Experimental and Therapeutic Medicine
Medicine
Cancer Research
Immunology
Microbiology
Sphingosine 1-Phosphate (S1P) Regulates Glucose-Stimulated Insulin Secretion in Pancreatic Beta Cells
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
