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Publications by Ivaylo Tournev

The Minus of a Plus Is a Minus. Mass Death of Selected Neuron Populations in Sporadic Late-Onset Neurodegenerative Disease May Be Due to a Combination of Subtly Decreased Capacity to Repair Oxidative DNA Damage and Increased Propensity for Damage-Related Apoptosis

Biotechnology and Biotechnological Equipment

Biotechnology

2016

English

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APOE4, Oxidative Stress and Decreased Repair Capacity - A No-Brainer. Faulty Lipid Metabolism and Increased Levels of Oxidative Damage May Be Risk Factors in the Pathogenesis of Late-Onset Dementia

BioDiscovery

2015

English

Repair of Oxidative Damage to Nuclear and Mitochondrial DNA in Mammalian Cells

Journal of Biological Chemistry

English

Correction for Degtyareva Et Al., “Chronic Oxidative DNA Damage Due to DNA Repair Defects Causes Chromosomal Instability in Saccharomyces Cerevisiae”

Molecular and Cellular Biology

Cell Biology

Molecular Biology

2019

English

Application of Hagedoorn’s Plus-Minus Method to Hydrology Study

2012

English

Slx4 Becomes Phosphorylated After DNA Damage in a Mec1/Tel1-Dependent Manner and Is Required for Repair of DNA Alkylation Damage

English

Resting T Cells Are Hypersensitive to DNA Damage Due to Defective DNA Repair Pathway

Cell Death and Disease

Molecular Neuroscience

English

Decreased Expression of the Augmenter of Liver Regeneration Results in Increased Apoptosis and Oxidative Damage in Human-Derived Glioma Cells

Cell Death and Disease

Molecular Neuroscience

English

Oxidative DNA Damage Background Estimated by a System Model of Base Excision Repair

Free Radical Biology and Medicine

Biochemistry

Physiology

2004

English

Oxidative Damage to DNA: Do We Have a Reliable Biomarker?

Environmental Health Perspectives

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by Ivaylo Tournev

The Minus of a Plus Is a Minus. Mass Death of Selected Neuron Populations in Sporadic Late-Onset Neurodegenerative Disease May Be Due to a Combination of Subtly Decreased Capacity to Repair Oxidative DNA Damage and Increased Propensity for Damage-Related Apoptosis

Related publications

APOE4, Oxidative Stress and Decreased Repair Capacity - A No-Brainer. Faulty Lipid Metabolism and Increased Levels of Oxidative Damage May Be Risk Factors in the Pathogenesis of Late-Onset Dementia

Repair of Oxidative Damage to Nuclear and Mitochondrial DNA in Mammalian Cells

Correction for Degtyareva Et Al., “Chronic Oxidative DNA Damage Due to DNA Repair Defects Causes Chromosomal Instability in Saccharomyces Cerevisiae”

Application of Hagedoorn’s Plus-Minus Method to Hydrology Study

Slx4 Becomes Phosphorylated After DNA Damage in a Mec1/Tel1-Dependent Manner and Is Required for Repair of DNA Alkylation Damage

Resting T Cells Are Hypersensitive to DNA Damage Due to Defective DNA Repair Pathway

Decreased Expression of the Augmenter of Liver Regeneration Results in Increased Apoptosis and Oxidative Damage in Human-Derived Glioma Cells

Oxidative DNA Damage Background Estimated by a System Model of Base Excision Repair

Oxidative Damage to DNA: Do We Have a Reliable Biomarker?