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Publications by J D Nardozzi
Inhibition of Apoptotic Bax Translocation to the Mitochondria Is a Central Function of Parkin
Cell Death and Disease
Molecular Neuroscience
Immunology
Cell Biology
Cancer Research
Cellular
Medicine
Related publications
Inhibition of Pro-Apoptotic BAX by a Noncanonical Interaction Mechanism
Molecular Cell
Cell Biology
Molecular Biology
DJ-1 Is Indispensable for the S-Nitrosylation of Parkin, Which Maintains Function of Mitochondria
Scientific Reports
Multidisciplinary
TOM22, a Core Component of the Mitochondria Outer Membrane Protein Translocation Pore, Is a Mitochondrial Receptor for the Proapoptotic Protein Bax
Cell Death and Differentiation
Cell Biology
Molecular Biology
BARD1 Translocation to Mitochondria Correlates With Bax Oligomerization, Loss of Mitochondrial Membrane Potential, and Apoptosis
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
BAX Mitochondrial Translocation Is Constitutive and Dynamic
Cancer Discovery
Oncology
Conformational Rearrangements in the Pro-Apoptotic Protein, Bax, as It Inserts Into Mitochondria
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Bax Function in the Absence of Mitochondria in the Primitive Protozoan Giardia Lamblia
PLoS ONE
Multidisciplinary
Apoptotic Response to Homoharringtonine in Human Wt P53 Leukemic Cells Is Independent of Reactive Oxygen Species Generation and Implicates Bax Translocation, Mitochondrial Cytochrome C Release and Caspase Activation
Leukemia
Cancer Research
Oncology
Anesthesiology
Pain Medicine
Hematology
High BCR-Abl Expression Prevents the Translocation of Bax and Bad to the Mitochondrion
Leukemia
Cancer Research
Oncology
Anesthesiology
Pain Medicine
Hematology
