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Publications by J M Zabolotny

Selection of a Nonconsensus Branch Point Is Influenced by an RNA Stem-Loop Structure and Is Important to Confer Stability to the Herpes Simplex Virus 2-Kilobase Latency-Associated Transcript.

Journal of Virology

English

Related publications

Analysis of the 2-Kilobase Latency-Associated Transcript Expressed in PC12 Cells Productively Infected With Herpes Simplex Virus Type 1: Evidence for a Stable, Nonlinear Structure.

English

A Protein Encoded by the Herpes Simplex Virus (HSV) Type 1 2-Kilobase Latency-Associated Transcript Is Phosphorylated, Localized to the Nucleus, and Overcomes the Repression of Expression From Exogenous Promoters When Inserted Into the Quiescent HSV Genome

English

Upstream Stimulatory Factor Family Binds to the Herpes Simplex Virus Type 1 Latency-Associated Transcript Promoter

Virology

1997

English

The Herpes Simplex Virus Type 1 Latency Associated Transcript Locus Is Required for the Maintenance of Reactivation Competent Latent Infections

Journal of NeuroVirology

Virology

Neurology

Cellular

Molecular Neuroscience

2011

English

The Latency-Associated Transcript Gene Enhances Establishment of Herpes Simplex Virus Type 1 Latency in Rabbits

English

Expression of the Herpes Simplex Virus Type 2 Latency-Associated Transcript Enhances Spontaneous Reactivation of Genital Herpes in Latently Infected Guinea Pigs

Journal of Experimental Medicine

English

A Herpes Simplex Virus Type 1 Latency-Associated Transcript Mutant With Increased Virulence and Reduced Spontaneous Reactivation

English

The Herpes Simplex Virus Type 1 Latency-Associated Transcript Promoter Is Activated Through Ras and Raf by Nerve Growth Factor and Sodium Butyrate in PC12 Cells.

English

Experimental Investigation of Herpes Simplex Virus Latency.

Clinical Microbiology Reviews

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by J M Zabolotny

Selection of a Nonconsensus Branch Point Is Influenced by an RNA Stem-Loop Structure and Is Important to Confer Stability to the Herpes Simplex Virus 2-Kilobase Latency-Associated Transcript.

Related publications

Analysis of the 2-Kilobase Latency-Associated Transcript Expressed in PC12 Cells Productively Infected With Herpes Simplex Virus Type 1: Evidence for a Stable, Nonlinear Structure.

A Protein Encoded by the Herpes Simplex Virus (HSV) Type 1 2-Kilobase Latency-Associated Transcript Is Phosphorylated, Localized to the Nucleus, and Overcomes the Repression of Expression From Exogenous Promoters When Inserted Into the Quiescent HSV Genome

Upstream Stimulatory Factor Family Binds to the Herpes Simplex Virus Type 1 Latency-Associated Transcript Promoter

The Herpes Simplex Virus Type 1 Latency Associated Transcript Locus Is Required for the Maintenance of Reactivation Competent Latent Infections

The Latency-Associated Transcript Gene Enhances Establishment of Herpes Simplex Virus Type 1 Latency in Rabbits

Expression of the Herpes Simplex Virus Type 2 Latency-Associated Transcript Enhances Spontaneous Reactivation of Genital Herpes in Latently Infected Guinea Pigs

A Herpes Simplex Virus Type 1 Latency-Associated Transcript Mutant With Increased Virulence and Reduced Spontaneous Reactivation

The Herpes Simplex Virus Type 1 Latency-Associated Transcript Promoter Is Activated Through Ras and Raf by Nerve Growth Factor and Sodium Butyrate in PC12 Cells.

Experimental Investigation of Herpes Simplex Virus Latency.