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Publications by J. F. Caro

Monoclonal Antibodies to the Human Insulin Receptor That Activate Glucose Transport but Not Insulin Receptor Kinase Activity.

Proceedings of the National Academy of Sciences of the United States of America

Multidisciplinary

1987

English

Related publications

Monoclonal Antibodies to the Insulin Receptor Mimic Metabolic Effects of Insulin but Do Not Stimulate Receptor Autophosphorylation in Transfected NIH 3T3 Fibroblasts.

Proceedings of the National Academy of Sciences of the United States of America

Multidisciplinary

1989

English

Protein Kinase Activity of the Insulin Receptor

English

Insulin Receptor Is an Insulin-Dependent Tyrosine Protein Kinase: Copurification of Insulin-Binding Activity and Protein Kinase Activity to Homogeneity From Human Placenta.

Proceedings of the National Academy of Sciences of the United States of America

Multidisciplinary

1984

English

Protein Kinase C Isoforms Α, Δ and Θ Require Insulin Receptor Substrate-1 to Inhibit the Tyrosine Kinase Activity of the Insulin Receptor in Human Kidney Embryonic Cells (HEK 293 Cells)

English

Evidence for an Insulin Receptor Substrate 1 Independent Insulin Signaling Pathway That Mediates Insulin-Responsive Glucose Transporter (GLUT4) Translocation.

Proceedings of the National Academy of Sciences of the United States of America

Multidisciplinary

1996

English

In Vivo Stimulation of the Insulin Receptor Kinase in Human Skeletal Muscle. Correlation With Insulin-Stimulated Glucose Disposal During Euglycemic Clamp Studies.

Journal of Clinical Investigation

Medicine

1991

English

Fructose, but Not Glucose, Impairs Insulin Signaling in the Three Major Insulin-Sensitive Tissues

Scientific Reports

Multidisciplinary

2016

English

The Drosophila Insulin Receptor Activates Multiple Signaling Pathways but Requires Insulin Receptor Substrate Proteins for DNA Synthesis.

Molecular and Cellular Biology

Cell Biology

Molecular Biology

1996

English

S-Nitrosation of the Insulin Receptor, Insulin Receptor Substrate 1, and Protein Kinase B/Akt: A Novel Mechanism of Insulin Resistance

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by J. F. Caro

Monoclonal Antibodies to the Human Insulin Receptor That Activate Glucose Transport but Not Insulin Receptor Kinase Activity.

Related publications

Monoclonal Antibodies to the Insulin Receptor Mimic Metabolic Effects of Insulin but Do Not Stimulate Receptor Autophosphorylation in Transfected NIH 3T3 Fibroblasts.

Protein Kinase Activity of the Insulin Receptor

Insulin Receptor Is an Insulin-Dependent Tyrosine Protein Kinase: Copurification of Insulin-Binding Activity and Protein Kinase Activity to Homogeneity From Human Placenta.

Protein Kinase C Isoforms Α, Δ and Θ Require Insulin Receptor Substrate-1 to Inhibit the Tyrosine Kinase Activity of the Insulin Receptor in Human Kidney Embryonic Cells (HEK 293 Cells)

Evidence for an Insulin Receptor Substrate 1 Independent Insulin Signaling Pathway That Mediates Insulin-Responsive Glucose Transporter (GLUT4) Translocation.

In Vivo Stimulation of the Insulin Receptor Kinase in Human Skeletal Muscle. Correlation With Insulin-Stimulated Glucose Disposal During Euglycemic Clamp Studies.

Fructose, but Not Glucose, Impairs Insulin Signaling in the Three Major Insulin-Sensitive Tissues

The Drosophila Insulin Receptor Activates Multiple Signaling Pathways but Requires Insulin Receptor Substrate Proteins for DNA Synthesis.

S-Nitrosation of the Insulin Receptor, Insulin Receptor Substrate 1, and Protein Kinase B/Akt: A Novel Mechanism of Insulin Resistance