Discover open access scientific publications

Search, annotate, share and cite publications

Publications by J. Krutzfeldt

Serine Residues 1177/78/82 of the Insulin Receptor Are Required for Substrate Phosphorylation but Not Autophosphorylation

Diabetes

English

Related publications

Regulation of Insulin Sensitivity by Serine/Threonine Phosphorylation of Insulin Receptor Substrate Proteins IRS1 and IRS2

English

Insulin Receptor Substrate Gene Polymorphisms Are Associated With Metabolic Syndrome but Not With Its Components

Journal of Diabetes Mellitus

2013

English

Monoclonal Antibodies to the Insulin Receptor Mimic Metabolic Effects of Insulin but Do Not Stimulate Receptor Autophosphorylation in Transfected NIH 3T3 Fibroblasts.

Proceedings of the National Academy of Sciences of the United States of America

Multidisciplinary

1989

English

Serine 302 Phosphorylation of Mouse Insulin Receptor Substrate 1 (IRS1) Is Dispensable for Normal Insulin Signaling and Feedback Regulation by Hepatic S6 Kinase

Journal of Biological Chemistry

English

Insulin and Hydrocortisone, but Not Triiodothyronine, Are Required for the Differentiation of Pig Preadipocytes in Primary Culture.

Journal of Animal Science

English

Insulin Stimulates PKCζ-mediated Phosphorylation of Insulin Receptor Substrate-1 (IRS-1)

Journal of Biological Chemistry

English

The Drosophila Insulin Receptor Activates Multiple Signaling Pathways but Requires Insulin Receptor Substrate Proteins for DNA Synthesis.

Molecular and Cellular Biology

Cell Biology

Molecular Biology

1996

English

Human Insulin Receptor Substrate-1 (IRS-1) Polymorphism G972R Causes IRS-1 to Associate With the Insulin Receptor and Inhibit Receptor Autophosphorylation

Journal of Biological Chemistry

English

Sphingomyelinase and Ceramide Suppress Insulin-Induced Tyrosine Phosphorylation of the Insulin Receptor Substrate-1

Journal of Biological Chemistry

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by J. Krutzfeldt

Serine Residues 1177/78/82 of the Insulin Receptor Are Required for Substrate Phosphorylation but Not Autophosphorylation

Related publications

Regulation of Insulin Sensitivity by Serine/Threonine Phosphorylation of Insulin Receptor Substrate Proteins IRS1 and IRS2

Insulin Receptor Substrate Gene Polymorphisms Are Associated With Metabolic Syndrome but Not With Its Components

Monoclonal Antibodies to the Insulin Receptor Mimic Metabolic Effects of Insulin but Do Not Stimulate Receptor Autophosphorylation in Transfected NIH 3T3 Fibroblasts.

Serine 302 Phosphorylation of Mouse Insulin Receptor Substrate 1 (IRS1) Is Dispensable for Normal Insulin Signaling and Feedback Regulation by Hepatic S6 Kinase

Insulin and Hydrocortisone, but Not Triiodothyronine, Are Required for the Differentiation of Pig Preadipocytes in Primary Culture.

Insulin Stimulates PKCζ-mediated Phosphorylation of Insulin Receptor Substrate-1 (IRS-1)

The Drosophila Insulin Receptor Activates Multiple Signaling Pathways but Requires Insulin Receptor Substrate Proteins for DNA Synthesis.

Human Insulin Receptor Substrate-1 (IRS-1) Polymorphism G972R Causes IRS-1 to Associate With the Insulin Receptor and Inhibit Receptor Autophosphorylation

Sphingomyelinase and Ceramide Suppress Insulin-Induced Tyrosine Phosphorylation of the Insulin Receptor Substrate-1