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Publications by J. Mushack
Protein Kinase C Isoforms Α, Δ and Θ Require Insulin Receptor Substrate-1 to Inhibit the Tyrosine Kinase Activity of the Insulin Receptor in Human Kidney Embryonic Cells (HEK 293 Cells)
Diabetologia
Internal Medicine
Endocrinology
Metabolism
Diabetes
Subcellular Distribution of GLUT 4 in the Skeletal Muscle of Lean Type 2 (Non-Insulin-Dependent) Diabetic Patients in the Basal State
Diabetologia
Internal Medicine
Endocrinology
Metabolism
Diabetes
Related publications
Insulin Receptor Is an Insulin-Dependent Tyrosine Protein Kinase: Copurification of Insulin-Binding Activity and Protein Kinase Activity to Homogeneity From Human Placenta.
Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
Protein Kinase C Directly Phosphorylates the Insulin Receptor in Vitro and Reduces Its Protein-Tyrosine Kinase Activity.
Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
Protein Kinase Activity of the Insulin Receptor
Biochemical Journal
Biochemistry
Cell Biology
Molecular Biology
Protein Kinase C-Ζ-Induced Phosphorylation of Ser318in Insulin Receptor Substrate-1 (IRS-1) Attenuates the Interaction With the Insulin Receptor and the Tyrosine Phosphorylation of IRS-1
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Serine Residues 994 and 1023/25 Are Important for Insulin Receptor Kinase Inhibition by Protein Kinase C Isoforms Β2 and Θ
Diabetologia
Internal Medicine
Endocrinology
Metabolism
Diabetes
S-Nitrosation of the Insulin Receptor, Insulin Receptor Substrate 1, and Protein Kinase B/Akt: A Novel Mechanism of Insulin Resistance
Diabetes
Internal Medicine
Endocrinology
Metabolism
Diabetes
Protein Kinase C in Insulin Releasing Cells
FEBS Letters
Genetics
Cell Biology
Molecular Biology
Biochemistry
Structural Biology
Biophysics
Human Insulin Receptor Substrate-1 (IRS-1) Polymorphism G972R Causes IRS-1 to Associate With the Insulin Receptor and Inhibit Receptor Autophosphorylation
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Insulin Receptor Substrate-2 Mediated Insulin-Like Growth Factor-I Receptor Overexpression in Pancreatic Adenocarcinoma Through Protein Kinase C
Cancer Research
Cancer Research
Oncology