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Publications by Jacob T. Williams
HDAC1 Is a Required Cofactor of CBFβ-SMMHC and a Potential Therapeutic Target in Inversion 16 Acute Myeloid Leukemia
Molecular Cancer Research
Cancer Research
Oncology
Molecular Biology
Related publications
Targeting Binding Partners of the CBFβ-SMMHC Fusion Protein for the Treatment of Inversion 16 Acute Myeloid Leukemia
Oncotarget
Oncology
Oncogenic CBFβ-SMMHC Fusion Protein Triggers Acute Leukemia in Conditional Mouse Knockin Model
Cancer Biology and Therapy
Cancer Research
Oncology
Molecular Medicine
Pharmacology
Multidrug Resistance Protein 4/ ATP Binding Cassette Transporter 4: A New Potential Therapeutic Target for Acute Myeloid Leukemia
Oncotarget
Oncology
Interleukin-3 Amplifies Acute Inflammation and Is a Potential Therapeutic Target in Sepsis
Science
Multidisciplinary
Philosophy of Science
History
Prosurvival Kinase PIM2 Is a Therapeutic Target for Eradication of Chronic Myeloid Leukemia Stem Cells
Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
Therapeutic Potential of GSK-J4, a Histone Demethylase KDM6B/JMJD3 Inhibitor, for Acute Myeloid Leukemia
Journal of Cancer Research and Clinical Oncology
Medicine
Cancer Research
Oncology
STAT3β Is a Tumor Suppressor in Acute Myeloid Leukemia
Intrinsic Activity
A Novel, Dual Pan-Pim/Flt3 Inhibitor SEL24 Exhibits Broad Therapeutic Potential in Acute Myeloid Leukemia
Oncotarget
Oncology
BCL-2 Is a Therapeutic Target for Hypodiploid B-Lineage Acute Lymphoblastic Leukemia
Cancer Research
Cancer Research
Oncology