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Publications by James E Vince
Mitochondrial Apoptosis Is Dispensable for NLRP3 Inflammasome Activation but Non-Apoptotic Caspase-8 Is Required for Inflammasome Priming
EMBO Reports
Biochemistry
Genetics
Molecular Biology
CD52 Inhibits Toll-Like Receptor Activation of NF-κB and Triggers Apoptosis to Suppress Inflammation
Cell Death and Differentiation
Cell Biology
Molecular Biology
Related publications
The GTPase Rab1 Is Required for NLRP3 Inflammasome Activation and Inflammatory Lung Injury
Journal of Immunology
Allergy
Immunology
HSP70 Is a Negative Regulator of NLRP3 Inflammasome Activation
Cell Death and Disease
Molecular Neuroscience
Immunology
Cell Biology
Cancer Research
Cellular
Medicine
NLRP3 Inflammasome Activation Is Associated With Proliferative Diabetic Retinopathy
Acta Ophthalmologica
Medicine
Ophthalmology
The Protein Kinase PKR Is Critical for LPS-induced iNOS Production but Dispensable for Inflammasome Activation in Macrophages
European Journal of Immunology
Allergy
Immunology
Caspase-3 Is Required for Α-Fodrin Cleavage but Dispensable for Cleavage of Other Death Substrates in Apoptosis
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Mitochondrial DNA Synthesis Fuels NLRP3 Inflammasome
Cell Research
Cell Biology
Molecular Biology
Age-Dependent Susceptibility to Pulmonary Fibrosis Is Associated With NLRP3 Inflammasome Activation
American Journal of Respiratory Cell and Molecular Biology
Respiratory Medicine
Pulmonary
Clinical Biochemistry
Cell Biology
Molecular Biology
NLRP3 Inflammasome Activation by Paracoccidioides Brasiliensis
PLoS Neglected Tropical Diseases
Pharmacology
Public Health
Environmental
Toxicology
Infectious Diseases
Pharmaceutics
Occupational Health
Interleukin 27 Is Increased in Carotid Atherosclerosis and Promotes NLRP3 Inflammasome Activation
PLoS ONE
Multidisciplinary
