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Publications by James E. Bowe
Nupr1 Deletion Protects Against Glucose Intolerance by Increasing Beta Cell Mass
Diabetologia
Internal Medicine
Endocrinology
Metabolism
Diabetes
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Capsazepine Protects Against Neuronal Injury Caused by Oxygen Glucose Deprivation by InhibitingIh
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Deletion of Cavin/PTRF Causes Global Loss of Caveolae, Dyslipidemia, and Glucose Intolerance
Cell Metabolism
Cell Biology
Molecular Biology
Physiology
Targeted Deletion of Insm2 in Mice Result in Reduced Insulin Secretion and Glucose Intolerance
Journal of Translational Medicine
Biochemistry
Medicine
Genetics
Molecular Biology
Rapamycin Induces Glucose Intolerance in Mice by Reducing Islet Mass, Insulin Content, and Insulin Sensitivity
Journal of Molecular Medicine
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Drug Discovery
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Disruption of Hepatic Leptin Signaling Protects Mice From Age- And Diet-Related Glucose Intolerance
Diabetes
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Spontaneous Physical Activity Protects Against Fat Mass Gain
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Peroxisome Proliferator-Activated Receptor Α (PPARα) Protects Against Oleate-Induced INS-1E Beta Cell Dysfunction by Preserving Carbohydrate Metabolism
Diabetologia
Internal Medicine
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Erratum. Genetic Disruption of Adenosine Kinase in Mouse Pancreatic Β-Cells Protects Against High-Fat Diet–Induced Glucose Intolerance. Diabetes 2017;66:1928–1938
Diabetes
Internal Medicine
Endocrinology
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CART Knock Out Mice Have Impaired Insulin Secretion and Glucose Intolerance, Altered Beta Cell Morphology and Increased Body Weight
Regulatory Peptides