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Publications by Jay Morgenstern
Tumor Necrosis Factor-Α Triggers Mucus Production in Airway Epithelium Through an IκB Kinase Β-Dependent Mechanism
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Related publications
Inactivation of IκB-kinase-β Dependent Genes in Airway Epithelium Reduces Tobacco Smoke Induced Acute Airway Inflammation
International Immunopharmacology
Allergy
Immunology
Pharmacology
Glutathione Depletion Down-Regulates Tumor Necrosis Factor Α-Induced NF-κB Activity via IκB Kinase-Dependent and -Independent Mechanisms
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Airway Epithelium and Mucus
American Journal of Respiratory Cell and Molecular Biology
Respiratory Medicine
Pulmonary
Clinical Biochemistry
Cell Biology
Molecular Biology
Fascin Is Involved in Tumor Necrosis Factor-Α-Dependent Production of MMP9 in Cholangiocarcinoma
Laboratory Investigation
Forensic Medicine
Pathology
Cell Biology
Molecular Biology
Esophageal Expression of Active IκB Kinase-Β in Mice Up-Regulates Tumor Necrosis Factor and Granulocyte-Macrophage Colony-Stimulating Factor, Promoting Inflammation and Angiogenesis
Gastroenterology
Hepatology
Gastroenterology
ENDOTOXIN EVOKES INTESTINAL PRODUCTION OF TUMOR NECROSIS FACTOR Α.• 309
Pediatric Research
Child Health
Pediatrics
Perinatology
Iron-Mediated H2O2Production as a Mechanism for Cell Type-Specific Inhibition of Tumor Necrosis Factor Α-Induced but Not Interleukin-1β-Induced IκB Kinase Complex/Nuclear Factor-κB Activation
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Ceramide Kinase Regulates the Production of Tumor Necrosis Factor Α (TNFα) via Inhibition of TNFα-converting Enzyme
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Prostaglandin I2 Is Responsible for Ameliorating Prostaglandin E2 Stress in Stimulating the Expression of Tumor Necrosis Factor Α in a Β-Amyloid Protein -Dependent Mechanism
Oncotarget
Oncology