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Publications by Jiachang Zhang

Protein Kinase C-Α Overexpression Stimulates Akt Activity and Suppresses Apoptosis Induced by Interleukin 3 Withdrawal

Oncogene

English

Related publications

GqProtein-induced Apoptosis Is Mediated by AKT Kinase Inhibition That Leads to Protein Kinase C-Induced C-Jun N-Terminal Kinase Activation

Journal of Biological Chemistry

English

Role of Akt and C-Jun N-Terminal Kinase 2 in Apoptosis Induced by Interleukin-4 Deprivation

Molecular Biology of the Cell

Cell Biology

Molecular Biology

1998

English

Receptor Interacting Protein 3 Suppresses Vascular Smooth Muscle Cell Growth by Inhibition of the Phosphoinositide 3-Kinase-Akt Axis

Journal of Biological Chemistry

English

Overexpression of Protein-Tyrosine Phosphatase-1b in Adipocytes Inhibits Insulin-Stimulated Phosphoinositide 3-Kinase Activity Without Altering Glucose Transport or Akt/Protein Kinase B Activation

Journal of Biological Chemistry

English

Inhibition of Phosphatidylinositol 3-Kinase by PX-866 Suppresses Temozolomide-Induced Autophagy and Promotes Apoptosis in Glioblastoma Cells

English

Inhibition of Protein Kinase C by Isojacareubin Suppresses Hepatocellular Carcinoma Metastasis and Induces Apoptosis in Vitro and in Vivo

Scientific Reports

Multidisciplinary

2015

English

Protein Kinase C Activity and Hexamethylenebisacetamide-Induced Erythroleukemia Cell Differentiation.

Proceedings of the National Academy of Sciences of the United States of America

Multidisciplinary

1987

English

Correlation Between Sustained C-Jun N-Terminal Protein Kinase Activation and Apoptosis Induced by Tumor Necrosis Factor-Α in Rat Mesangial Cells

Journal of Biological Chemistry

English

Nuclear Protein Kinase C Isoforms and Apoptosis

European Journal of Histochemistry

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by Jiachang Zhang

Protein Kinase C-Α Overexpression Stimulates Akt Activity and Suppresses Apoptosis Induced by Interleukin 3 Withdrawal

Related publications

GqProtein-induced Apoptosis Is Mediated by AKT Kinase Inhibition That Leads to Protein Kinase C-Induced C-Jun N-Terminal Kinase Activation

Role of Akt and C-Jun N-Terminal Kinase 2 in Apoptosis Induced by Interleukin-4 Deprivation

Receptor Interacting Protein 3 Suppresses Vascular Smooth Muscle Cell Growth by Inhibition of the Phosphoinositide 3-Kinase-Akt Axis

Overexpression of Protein-Tyrosine Phosphatase-1b in Adipocytes Inhibits Insulin-Stimulated Phosphoinositide 3-Kinase Activity Without Altering Glucose Transport or Akt/Protein Kinase B Activation

Inhibition of Phosphatidylinositol 3-Kinase by PX-866 Suppresses Temozolomide-Induced Autophagy and Promotes Apoptosis in Glioblastoma Cells

Inhibition of Protein Kinase C by Isojacareubin Suppresses Hepatocellular Carcinoma Metastasis and Induces Apoptosis in Vitro and in Vivo

Protein Kinase C Activity and Hexamethylenebisacetamide-Induced Erythroleukemia Cell Differentiation.

Correlation Between Sustained C-Jun N-Terminal Protein Kinase Activation and Apoptosis Induced by Tumor Necrosis Factor-Α in Rat Mesangial Cells

Nuclear Protein Kinase C Isoforms and Apoptosis