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Publications by Jin Bo Feng

Cross Talk Among Smad, MAPK, and Integrin Signaling Pathways Enhances Adventitial Fibroblast Functions Activated by Transforming Growth Factor- 1 and Inhibited by Gax

Arteriosclerosis, Thrombosis, and Vascular Biology
Cardiovascular MedicineCardiology
2008English

Related publications

TGF-beta1 Stimulates Human AT1 Receptor Expression in Lung Fibroblasts by Cross Talk Between the Smad, P38 MAPK, JNK, and PI3K Signaling Pathways

American Journal of Physiology - Lung Cellular and Molecular Physiology
PulmonaryRespiratory MedicinePhysiologyCell Biology
2007English

Nitric Oxide Induces TIMP-1 Expression by Activating the Transforming Growth Factor Β-Smad Signaling Pathway

Journal of Biological Chemistry
BiochemistryCell BiologyMolecular Biology
2005English

BCL6 Represses Smad Signaling in Transforming Growth Factor- Resistance

Cancer Research
Cancer ResearchOncology
2008English

Endogenous Transforming Growth Factor-Β Receptor-Mediated Smad Signaling Complexes Analyzed by Mass Spectrometry

Molecular and Cellular Proteomics
BiochemistryMedicineAnalytical ChemistryMolecular Biology
2006English

Receptor Type Protein Tyrosine Phosphatase-Kappa Mediates Cross-Talk Between Transforming Growth Factor-Beta and Epidermal Growth Factor Receptor Signaling Pathways in Human Keratinocytes

Molecular Biology of the Cell
Cell BiologyMolecular Biology
2010English

Both the Smad and P38 MAPK Pathways Play a Crucial Role in Runx2 Expression Following Induction by Transforming Growth Factor-Β and Bone Morphogenetic Protein

Oncogene
Cancer ResearchGeneticsMolecular Biology
2002English

Inactivation of Smad-Transforming Growth Factor Beta Signaling by Ca2+-Calmodulin-Dependent Protein Kinase II

Molecular and Cellular Biology
Cell BiologyMolecular Biology
2000English

Modulation of the Transforming Growth Factor-Β1-Induced Smad Phosphorylation by the Extracellular Matrix Receptor Β1-Integrin

International Journal of Oncology
Cancer ResearchOncology
2009English

Positive and Negative Modulation of Vitamin D Receptor Function by Transforming Growth Factor-Β Signaling Through Smad Proteins

Journal of Biological Chemistry
BiochemistryCell BiologyMolecular Biology
1999English

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