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Publications by JoEllen Fabritz

Mutations That Induce Constitutive Activation and Mutations That Impair Signal Transduction Modulate the Basal and/or Agonist-Stimulated Internalization of the Lutropin/Choriogonadotropin Receptor

Journal of Biological Chemistry

English

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An Opioid Agonist That Does Not Induce -Opioid Receptor--Arrestin Interactions or Receptor Internalization

Molecular Pharmacology

Molecular Medicine

Pharmacology

2006

English

Effects of Second Intracellular Loop Mutations on Signal Transduction and Internalization of the Gonadotropin-Releasing Hormone Receptor

Journal of Biological Chemistry

English

Mutation of Individual Serine Residues in the C-Terminal Tail of the Lutropin/Choriogonadotropin Receptor Reveal Distinct Structural Requirements for Agonist-Induced Uncoupling and Agonist-Induced Internalization

Journal of Biological Chemistry

English

A Model for Constitutive Lutropin Receptor Activation Based on Molecular Simulation and Engineered Mutations in Transmembrane Helices 6 and 7

Journal of Biological Chemistry

English

Factors That Effect Signal Transduction by the Estrogen Receptor.

1997

English

Constitutive Activation of the Δ Opioid Receptor by Mutations in Transmembrane Domains III and VII

Journal of Biological Chemistry

English

Constitutive Activation and Uncoupling of the Atrial Natriuretic Peptide Receptor by Mutations at the Dimer Interface

Journal of Biological Chemistry

English

The Association of Arrestin-3 With the Human Lutropin/Choriogonadotropin Receptor Depends Mostly on Receptor Activation Rather Than on Receptor Phosphorylation

Journal of Biological Chemistry

English

Mutations Affecting the Glycine Receptor Agonist Transduction Mechanism Convert the Competitive Antagonist, Picrotoxin, Into an Allosteric Potentiator

Journal of Biological Chemistry

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by JoEllen Fabritz

Mutations That Induce Constitutive Activation and Mutations That Impair Signal Transduction Modulate the Basal and/or Agonist-Stimulated Internalization of the Lutropin/Choriogonadotropin Receptor

Related publications

An Opioid Agonist That Does Not Induce -Opioid Receptor--Arrestin Interactions or Receptor Internalization

Effects of Second Intracellular Loop Mutations on Signal Transduction and Internalization of the Gonadotropin-Releasing Hormone Receptor

Mutation of Individual Serine Residues in the C-Terminal Tail of the Lutropin/Choriogonadotropin Receptor Reveal Distinct Structural Requirements for Agonist-Induced Uncoupling and Agonist-Induced Internalization

A Model for Constitutive Lutropin Receptor Activation Based on Molecular Simulation and Engineered Mutations in Transmembrane Helices 6 and 7

Factors That Effect Signal Transduction by the Estrogen Receptor.

Constitutive Activation of the Δ Opioid Receptor by Mutations in Transmembrane Domains III and VII

Constitutive Activation and Uncoupling of the Atrial Natriuretic Peptide Receptor by Mutations at the Dimer Interface

The Association of Arrestin-3 With the Human Lutropin/Choriogonadotropin Receptor Depends Mostly on Receptor Activation Rather Than on Receptor Phosphorylation

Mutations Affecting the Glycine Receptor Agonist Transduction Mechanism Convert the Competitive Antagonist, Picrotoxin, Into an Allosteric Potentiator