Discover open access scientific publications

Search, annotate, share and cite publications

Publications by John J Kopchick

Mitochondrial Function Is Compromised in Cortical Bone Osteocytes of Long-Lived Growth Hormone Receptor Null Mice

Journal of Bone and Mineral Research

English

MECHANISMS IN ENDOCRINOLOGY: Lessons From Growth Hormone Receptor Gene Disrupted Mice: Are There Benefits of Endocrine Defects?

European Journal of Endocrinology

English

Related publications

Altered Structure and Function of Adipose Tissue in Long-Lived Mice With Growth Hormone-Related Mutations

English

Growth Hormone Abolishes Beneficial Effects of Calorie Restriction in Long-Lived Ames Dwarf Mice

Experimental Gerontology

English

Mechanical and Material Properties of Cortical and Trabecular Bone From Cannabinoid Receptor-1-Null (Cnr1−/−) Mice

Medical Engineering and Physics

Biomedical Engineering

Biophysics

2016

English

Decline in the Growth Potential of Spleen-Colonizing Bone Marrow Stem Cells of Long-Lived Aging Mice

Journal of Experimental Medicine

English

Growth Hormone Effects on Cortical Bone Dimensions in Young Adults With Childhood-Onset Growth Hormone Deficiency

Osteoporosis International

English

Notch Signaling in Osteocytes Differentially Regulates Cancellous and Cortical Bone Remodeling

Journal of Biological Chemistry

English

The D152H Mutation Found in Growth Hormone Insensitivity Syndrome Impairs Expression and Function of Human Growth Hormone Receptor but Is Silent in Rat Receptor

Journal of Molecular Endocrinology

Endocrinology

Molecular Biology

1998

English

Osteoblast Function Is Compromised at Sites of Focal Bone Erosion in Inflammatory Arthritis

Journal of Bone and Mineral Research

English

Up-Regulation of Thrombospondin-2 in Akt1-Null Mice Contributes to Compromised Tissue Repair Due to Abnormalities in Fibroblast Function

Journal of Biological Chemistry

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by John J Kopchick

Mitochondrial Function Is Compromised in Cortical Bone Osteocytes of Long-Lived Growth Hormone Receptor Null Mice

MECHANISMS IN ENDOCRINOLOGY: Lessons From Growth Hormone Receptor Gene Disrupted Mice: Are There Benefits of Endocrine Defects?

Related publications

Altered Structure and Function of Adipose Tissue in Long-Lived Mice With Growth Hormone-Related Mutations

Growth Hormone Abolishes Beneficial Effects of Calorie Restriction in Long-Lived Ames Dwarf Mice

Mechanical and Material Properties of Cortical and Trabecular Bone From Cannabinoid Receptor-1-Null (Cnr1−/−) Mice

Decline in the Growth Potential of Spleen-Colonizing Bone Marrow Stem Cells of Long-Lived Aging Mice

Growth Hormone Effects on Cortical Bone Dimensions in Young Adults With Childhood-Onset Growth Hormone Deficiency

Notch Signaling in Osteocytes Differentially Regulates Cancellous and Cortical Bone Remodeling

The D152H Mutation Found in Growth Hormone Insensitivity Syndrome Impairs Expression and Function of Human Growth Hormone Receptor but Is Silent in Rat Receptor

Osteoblast Function Is Compromised at Sites of Focal Bone Erosion in Inflammatory Arthritis

Up-Regulation of Thrombospondin-2 in Akt1-Null Mice Contributes to Compromised Tissue Repair Due to Abnormalities in Fibroblast Function