Discover open access scientific publications

Search, annotate, share and cite publications

Publications by John Savory

Co-Involvement of Mitochondria and Endoplasmic Reticulum in Regulation of Apoptosis: Changes in Cytochrome C, BCL-2 and Bax in the Hippocampus of Aluminum-Treated Rabbits

Brain Research

Neuroscience

Neurology

Developmental Biology

Molecular Biology

2001

English

1582 Aluminum Hydroxide, Vitamin D, Dihydrotachysterol (Dht), 1,25-Dihydroxyvitamin-D (1,25-D) on Aluminum Excretion in Rats

English

Related publications

Tubeimoside-1 Induces Glioma Apoptosis Through Regulation of Bax/BCL-2 and the ROS/Cytochrome C/Caspase-3 Pathway

OncoTargets and Therapy

Oncology

Pharmacology

2015

English

The Endoplasmic Reticulum in Apoptosis and Autophagy: Role of the BCL-2 Protein Family

English

Involvement of BNIP1 in Apoptosis and Endoplasmic Reticulum Membrane Fusion

English

Role of Mitochondria and Endoplasmic Reticulum in Taurine-Deficiency-Mediated Apoptosis

English

Apoptosis in Normal Rat Embryo Tissues During Early Organogenesis: The Possible Involvement of Bax and BCL-2.

Archives of Histology and Cytology

Histology

2002

English

Bax and Bak Can Localize to the Endoplasmic Reticulum to Initiate Apoptosis

Journal of Cell Biology

Medicine

Cell Biology

2003

English

Endoplasmic Reticulum Mitochondria Contacts Modulate Apoptosis of Renal Cells and Its Implications in Diabetic Neuropathy

English

Growth Factors Prevent Changes in BCL-2 and Bax Expression and Neuronal Apoptosis Induced by Nitric Oxide

Cell Death and Differentiation

Cell Biology

Molecular Biology

1998

English

Lipopolysaccharide-Induced Dental Pulp Cell Apoptosis and the Expression of Bax and BCL-2 in Vitro

Brazilian Journal of Medical and Biological Research

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by John Savory

Co-Involvement of Mitochondria and Endoplasmic Reticulum in Regulation of Apoptosis: Changes in Cytochrome C, BCL-2 and Bax in the Hippocampus of Aluminum-Treated Rabbits

1582 Aluminum Hydroxide, Vitamin D, Dihydrotachysterol (Dht), 1,25-Dihydroxyvitamin-D (1,25-D) on Aluminum Excretion in Rats

Related publications

Tubeimoside-1 Induces Glioma Apoptosis Through Regulation of Bax/BCL-2 and the ROS/Cytochrome C/Caspase-3 Pathway

The Endoplasmic Reticulum in Apoptosis and Autophagy: Role of the BCL-2 Protein Family

Involvement of BNIP1 in Apoptosis and Endoplasmic Reticulum Membrane Fusion

Role of Mitochondria and Endoplasmic Reticulum in Taurine-Deficiency-Mediated Apoptosis

Apoptosis in Normal Rat Embryo Tissues During Early Organogenesis: The Possible Involvement of Bax and BCL-2.

Bax and Bak Can Localize to the Endoplasmic Reticulum to Initiate Apoptosis

Endoplasmic Reticulum Mitochondria Contacts Modulate Apoptosis of Renal Cells and Its Implications in Diabetic Neuropathy

Growth Factors Prevent Changes in BCL-2 and Bax Expression and Neuronal Apoptosis Induced by Nitric Oxide

Lipopolysaccharide-Induced Dental Pulp Cell Apoptosis and the Expression of Bax and BCL-2 in Vitro