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Publications by Jonathan C. Kagan
A Single Bacterial Immune Evasion Strategy Dismantles Both MyD88 and TRIF Signaling Pathways Downstream of TLR4
Cell Host and Microbe
Immunology
Molecular Biology
Cancer Research
Virology
Microbiology
Microbiology
Parasitology
Related publications
Steady-State Neutrophil Homeostasis Is Dependent on TLR4/TRIF Signaling
Blood
Biochemistry
Immunology
Cell Biology
Hematology
BZS1, a B-Box Protein, Promotes Photomorphogenesis Downstream of Both Brassinosteroid and Light Signaling Pathways
Molecular Plant
Plant Science
Molecular Biology
Pepsin Digest of Wheat Gliadin Fraction Increases Production of IL-1β via TLR4/MyD88/TRIF/MAPK/NF-κB Signaling Pathway and an NLRP3 Inflammasome Activation
PLoS ONE
Multidisciplinary
Mycobacterial Phenolic Glycolipids Selectively Disable TRIF-Dependent TLR4 Signaling in Macrophages
Frontiers in Immunology
Allergy
Immunology
Bacterial Evasion of the Host Immune System
Downstream Signals for MyD88-Mediated Phagocytosis of Borrelia Burgdorferi Can Be Initiated by TRIF and Are Dependent on PI3K
Journal of Immunology
Allergy
Immunology
HPV-transformed Cells Exhibit Altered HMGB1-TLR4/MyD88-SARM1 Signaling Axis
Scientific Reports
Multidisciplinary
Chondrocyte Innate Immune MyD88-dependent Signaling Drives Pro-Catabolic Effects of the Endogenous TLR2/TLR4 Ligands LMW-HA and HMGB1
Arthritis & Rheumatism
Mechanism of Focal Cerebral Ischemic Tolerance in Rats With Ischemic Preconditioning Involves MyD88- And TRIF-dependent Pathways
Experimental and Therapeutic Medicine
Medicine
Cancer Research
Immunology
Microbiology