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Publications by Joo Mi Yi
Aberrantly Hypermethylated Tumor Suppressor Genes Were Identified in Oral Squamous Cell Carcinoma (OSCC)
Clinical Epigenetics
Genetics
Developmental Biology
Molecular Biology
Eckol Inhibits Particulate Matter 2.5-Induced Skin Keratinocyte Damage via MAPK Signaling Pathway
Marine Drugs
Drug Discovery
Related publications
S100A16 Functions as a Tumor Suppressor Protein in Oral Squamous Cell Carcinoma
International Journal of Radiation Oncology Biology Physics
Oncology
Radiology
Cancer Research
Radiation
Nuclear Medicine
Imaging
Tropomyosin-1 Acts as a Potential Tumor Suppressor in Human Oral Squamous Cell Carcinoma
PLoS ONE
Multidisciplinary
Analysis of Expression of P53 Tumor Suppressor Gene in Human Oral Squamous Cell Carcinoma.
Kokubyo Gakkai zasshi. The Journal of the Stomatological Society, Japan
Medicine
MiR-1254 Functions as a Tumor Suppressor in Oral Squamous Cell Carcinoma by Targeting CD36
Technology in Cancer Research and Treatment
Medicine
Cancer Research
Oncology
C-Myc Expression in T (III/IV) Stage Oral Squamous Cell Carcinoma (OSCC) Patients
Cancer Management and Research
Oncology
Expression of the Tumor Suppressor Gene Hypermethylated in Cancer�1 in Laryngeal Carcinoma
Oncology Letters
Cancer Research
Oncology
MicroRNA-16 Functions as a Tumor-Suppressor Gene in Oral Squamous Cell Carcinoma by Targeting AKT3 and BCL2L2
Journal of Cellular Physiology
Clinical Biochemistry
Cell Biology
Physiology
MiR-206 Functions as a Tumor Suppressor and Directly Targets K-Ras in Human Oral Squamous Cell Carcinoma [Retraction]
OncoTargets and Therapy
Oncology
Pharmacology
ECRG4 Is a Candidate Tumor Suppressor Gene Frequently Hypermethylated in Colorectal Carcinoma and Glioma
BMC Cancer
Cancer Research
Oncology
Genetics