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Publications by Jorge Matias Caviglia
MicroRNA-21 and Dicer Are Dispensable for Hepatic Stellate Cell Activation and the Development of Liver Fibrosis
Hepatology
Medicine
Hepatology
Different Fatty Acids Inhibit apoB100 Secretion by Different Pathways: Unique Roles for ER Stress, Ceramide, and Autophagy
Journal of Lipid Research
Biochemistry
Endocrinology
Cell Biology
Related publications
Suppressive Effect of Orthovanadate on Hepatic Stellate Cell Activation and Liver Fibrosis in Rats
American Journal of Pathology
Forensic Medicine
Pathology
SRC Inhibition Attenuates Liver Fibrosis by Preventing Hepatic Stellate Cell Activation and Decreasing Connetive Tissue Growth Factor
Cells
Ferulic Acid Attenuates Liver Fibrosis and Hepatic Stellate Cell Activation via Inhibition of TGF-β/Smad Signaling Pathway
Drug Design, Development and Therapy
Drug Discovery
Pharmacology
Pharmaceutical Science
The Role of MicroRNA-21 and Autophagy in Liver Fibrosis
Journal of Liver
P53-Dependent Induction of Ferroptosis Is Required for Artemether to Alleviate Carbon Tetrachloride-Induced Liver Fibrosis and Hepatic Stellate Cell Activation
IUBMB Life
Biochemistry
Genetics
Clinical Biochemistry
Cell Biology
Molecular Biology
The Autoregulatory Feedback Loop of MicroRNA-21/Programmed Cell Death Protein 4/Activation Protein-1 (MiR-21/PDCD4/AP-1) as a Driving Force for Hepatic Fibrosis Development
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Hepatic Stellate Cell Activation and Pro-Fibrogenic Signals
Journal of Hepatology
Hepatology
NPAS2 Contributes to Liver Fibrosis by Directly Transcriptional Activation of Hes1 in Hepatic Stellate Cells
Molecular Therapy - Nucleic Acids
Drug Discovery
Molecular Medicine
Fluorofenidone Affects Hepatic Stellate Cell Activation in Hepatic Fibrosis by Targeting the TGF‑β1/Smad and MAPK Signaling Pathways
Experimental and Therapeutic Medicine
Medicine
Cancer Research
Immunology
Microbiology