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Publications by Josef Klappacher
MEIS2 Is an Oncogenic Partner in AML1-ETO-Positive AML
Cell Reports
Biochemistry
Genetics
Molecular Biology
Related publications
A Histone Acetyltransferase P300 Inhibitor C646 Induces Cell Cycle Arrest and Apoptosis Selectively in AML1-ETO-Positive AML Cells
PLoS ONE
Multidisciplinary
NHR4 Domain Mutations of ETO Are Probably Very Infrequent in AML1–ETO Positive Myeloid Leukemia Cells
Leukemia
Cancer Research
Oncology
Anesthesiology
Pain Medicine
Hematology
Detection of DNA Rearrangements in the AML1 and ETO Loci and of an AML1/ETO Fusion mRNA in Patients With T(8;21) Acute Myeloid Leukemia
Blood
Biochemistry
Immunology
Cell Biology
Hematology
Targeting the Oligomerization Domain of ETO Interferes With RUNX1/ETO Oncogenic Activity in T(8;21)-Positive Leukemic Cells
Cancer Research
Cancer Research
Oncology
The Leukemogenicity of AML1-ETO Is Dependent on Site-Specific Lysine Acetylation
Science
Multidisciplinary
Philosophy of Science
History
Novel Role of HDAC Inhibitors in AML1/ETO AML Cells: Activation of Apoptosis and Phagocytosis Through Induction of Annexin A1
Cell Death and Differentiation
Cell Biology
Molecular Biology
Gene Mutation Profile and Risk Stratification in AML1‑ETO‑positive Acute Myeloid Leukemia Based on Next‑generation Sequencing
Oncology Reports
Medicine
Cancer Research
Oncology
Significance of the TEL-AML1 Fusion Gene in Childhood AML
Leukemia
Cancer Research
Oncology
Anesthesiology
Pain Medicine
Hematology
ETO, but Not Leukemogenic Fusion Protein AML1/ETO, Augments RBP-J /SHARP-Mediated Repression of Notch Target Genes
Molecular and Cellular Biology
Cell Biology
Molecular Biology
