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Publications by Joseph F Nabhan

Arrestin Domain-Containing Protein 3 Recruits the NEDD4 E3 Ligase to Mediate Ubiquitination of the Β2-Adrenergic Receptor

Molecular Biology

English

Related publications

Β-Arrestin–biased Signaling Through the Β2-Adrenergic Receptor Promotes Cardiomyocyte Contraction

Proceedings of the National Academy of Sciences of the United States of America

Multidisciplinary

English

The Nedd4-Binding Partner 1 (N4BP1) Protein Is an Inhibitor of the E3 Ligase Itch

Proceedings of the National Academy of Sciences of the United States of America

Multidisciplinary

English

Crystal Structure of the Β2 Adrenergic Receptor–Gs Protein Complex

Multidisciplinary

English

The E3 Ligase TRAF6 Regulates Akt Ubiquitination and Activation

Multidisciplinary

Philosophy of Science

English

The 3′-Untranslated Region of the Β2-Adrenergic Receptor mRNA Regulates Receptor Synthesis

Journal of Biological Chemistry

Molecular Biology

English

A Role for the E3 Ubiquitin Ligase NEDD4 in Keloid Pathogenesis

Journal of Investigative Dermatology

Molecular Biology

English

Β-Arrestin Is Crucial for Ubiquitination and Down-Regulation of the Insulin-Like Growth Factor-1 Receptor by Acting as Adaptor for the MDM2 E3 Ligase

Journal of Biological Chemistry

Molecular Biology

English

The E3 Ligase TTC3 Facilitates Ubiquitination and Degradation of Phosphorylated Akt

Developmental Cell

Developmental Biology

Molecular Biology

English

Conformational Changes in the G Protein Gs Induced by the Β2 Adrenergic Receptor

Multidisciplinary

English