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Publications by Junsheng Tian

Increasing the Level of IRS-1 and Insulin Pathway Sensitivity by Natural Product Carainterol A

Molecules
Organic ChemistryMolecular MedicineAnalytical ChemistryTheoretical ChemistryPharmaceutical ScienceMedicineDrug DiscoveryChemistryPhysical
2016English

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Mammary Tumorigenesis and Metastasis Caused by Overexpression of Insulin Receptor Substrate 1 (IRS-1) or IRS-2

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Regulation of Insulin Signalling by Hyperinsulinaemia: Role of IRS-1/2 Serine Phosphorylation and the mTOR/p70 S6K Pathway

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Human Insulin Receptor Substrate-1 (IRS-1) Polymorphism G972R Causes IRS-1 to Associate With the Insulin Receptor and Inhibit Receptor Autophosphorylation

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Exercise-Induced Muscle Damage Impairs Insulin Signaling Pathway Associated With IRS-1 Oxidative Modification

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Role of IRS-3 in the Insulin Signaling of IRS-1-deficient Brown Adipocytes

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Insulin Stimulates PKCζ-mediated Phosphorylation of Insulin Receptor Substrate-1 (IRS-1)

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Protein Kinase C-Ζ-Induced Phosphorylation of Ser318in Insulin Receptor Substrate-1 (IRS-1) Attenuates the Interaction With the Insulin Receptor and the Tyrosine Phosphorylation of IRS-1

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AMPK-TBC1D4–Dependent Mechanism for Increasing Insulin Sensitivity of Skeletal Muscle: Figure 1

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