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Publications by Juraj Bies
Oncogenic Activation of C-Myb by Carboxyl-Terminal Truncation Leads to Decreased Proteolysis by the Ubiquitin-26s Proteasome Pathway
Oncogene
Cancer Research
Genetics
Molecular Biology
Hypermethylation of the Ink4b Locus in Murine Myeloid Leukemia and Increased Susceptibility to Leukemia in P15Ink4b-Deficient Mice
Oncogene
Cancer Research
Genetics
Molecular Biology
Related publications
Correction To: Proteomic Analysis of Degradation Ubiquitin Signaling by Ubiquitin Occupancy Changes Responding to 26S Proteasome Inhibition
Clinical Proteomics
Molecular Medicine
Clinical Biochemistry
Molecular Biology
Hsp70–Hsp110 Chaperones Deliver Ubiquitin-Dependent and -Independent Substrates to the 26S Proteasome for Proteolysis in Yeast
Journal of Cell Science
Cell Biology
Inherent Asymmetry in the 26S Proteasome Is Defined by the Ubiquitin Receptor RPN13
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Elevated Expression of ISG15 in Tumor Cells Interferes With the Ubiquitin/26s Proteasome Pathway
Cancer Research
Cancer Research
Oncology
Drosophila Argonaute2 Turnover Is Regulated by the Ubiquitin Proteasome Pathway
Biochemical and Biophysical Research Communications
Biochemistry
Cell Biology
Molecular Biology
Biophysics
Proteolysis, the Ubiquitin-Proteasome System, and Renal Diseases
American Journal of Physiology - Renal Physiology
Urology
Physiology
Livin Promotes Smac/Diablo Degradation by Ubiquitin–proteasome Pathway
Cell Death and Differentiation
Cell Biology
Molecular Biology
C-Jun N-Terminal Kinase Activation by Nitrobenzoxadiazoles Leads to Late-Stage Autophagy Inhibition
Journal of Translational Medicine
Biochemistry
Medicine
Genetics
Molecular Biology
Loss of 26S Proteasome Function Leads to Increased Cell Size and Decreased Cell Number in Arabidopsis Shoot Organs
Plant Physiology
Plant Science
Genetics
Physiology